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Structural and Electrical Myocardial Remodeling in a Rodent Model of Depression

Bibliographic Data

ID19308564
AuthorsLuca Carnevali (0000-0001-9053-7976, University of Parma), Mimosa Trombini (University of Parma), Stefano Rossi (0000-0003-1692-5342, University of Groningen), Gallia Graiani (0000-0003-1926-7568, University of Groningen), Massimo Manghi (0000-0001-6413-3046, University of Groningen), Jaap M Koolhaas (University of Groningen), Federico Quaini (0000-0003-2377-4810, University of Parma), Emilio Macchi (0000-0002-8993-3987, University of Parma), Eugene Nalivaiko (0000-0002-6449-8181, University of Groningen), Andrea Sgoifo (0000-0002-9428-6198, University of Groningen, corresponding author)
Year2013
Volume75
Issue1
Pages42-51
Publication date2013-01-01
Peer ReviewedYes
Open AccessNo
TypeARTICLE
VenuePsychosomatic Medicine (JOURNAL)
Journal identifiersISSN: 0033-3174 • E-ISSN: 1534-7796
PublisherOvid Technologies (Wolters Kluwer Health) (PUBLISHER)
DOI10.1097/psy.0b013e318276cb0d
PMID23257930
OpenAlexW2148166641
LanguageEN
Citations received1
References cited27

OBJECTIVE: Despite a well-documented association between stress and depression with cardiac morbidity and mortality, there is no satisfactory explanation for the mechanisms linking affective and cardiac disorders. This study investigated cardiac electrophysiological properties in an animal model of depression. METHODS: Depression-relevant physiological and behavioral parameters were measured in adult male wild-type rats during and after a period of intermittent social defeat stress (n = 12) or empty cage exposure (control, n = 11). Nine days after the last defeat/empty cage exposure, high-definition epicardial mapping was performed under anesthesia. RESULTS: Stressed animals versus controls displayed a larger reduction in the circadian amplitude of heart rate (-32% [3%] versus -13 [2%]; p = .001) and body temperature (-33% [4%] versus -5% [2%]; p = .001) rhythms, had smaller body weight gain (+11% [1%] versus +17% [1%]; p < .001), and showed a larger reduction in sucrose solution intake (-19% [6%] versus -7% [4%]; p = .006). Epicardial mapping analysis revealed a decrease in the transversal conduction velocity of the wavefront (0.23 [0.0] versus 0.27 [0.1] m/s; p = .02) and a shortening of the effective refractory period (86.8 [2.1] versus 95.9 [3.0] milliseconds; p = .01) in stressed animals. Upon killing, moderate left ventricular fibrosis was observed in the stressed group. CONCLUSIONS: Intermittent social stress procedure is associated with depression-like symptoms and altered myocardial electrical stability in a potentially proarrhythmic manner. In particular, reduced myocardial refractoriness and impaired conduction, which are considered major determinants of arrhythmogenesis, represent possible mechanisms underlying cardiac vulnerability

Anesthesia · Cardiac Electrophysiology · Cardiology · Depression (economics) · Effective refractory period · Electrophysiology · Nerve conduction velocity · Refractory period · Cardiac electrophysiology and arrhythmias · Cardiac Fibrosis and Remodeling · Endocrinology · Internal Medicine · Medicine · Stress Responses and Cortisol

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Unique citing works1
Citations per year0,11
Citation span2017 - 2017 (1)
Citation velocityhistorical
Highly citedNo
Citation typesNeutral: 1
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