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The inhibitory effects of modified HSJZ decoction on NSCLC by regulating regulatory T cells via downregulation of EZH2 and PI3K/AKT pathway

Datos Bibliográficos

ID14729555
AutoresLuo Kewang (Third Affiliated Hospital of Southern Medical University, autor de correspondencia), Wei Ye (0000-0001-7484-0609, Southern Medical University), Ye Wei (0000-0001-8931-0926), Li Meiye (Southern Medical University), Hu Tianyong (Longgang Central Hospital), Cheng Baohui, Bao‐Hui Cheng (0000-0002-5065-9036, Longgang Central Hospital, autor de correspondencia)
Año2025
Volumen348
Páginas119802
Fecha de publicación2025-05-01
Peer ReviewedSí
Open AccessSí
TipoARTICLE
RevistaJournal of Ethnopharmacology (JOURNAL)
Identificadores de la revistaISSN: 0378-8741 • E-ISSN: 1872-7573
EditorialElsevier BV (PUBLISHER)
DOI10.1016/j.jep.2025.119802
PMID40245962
OpenAlexW4409449277
IdiomaEN
Referencias citadas35

These findings provide compelling evidence that HSJZ exerts anti-tumor effects in NSCLC by modulating the immune microenvironment, particularly through the inhibition of Tregs activity, as assessed by in vitro co-culture system, in vivo mouse orthotopic Lewis cancer model, immunohistochemistry, etc. Our results support the potential clinical application of HSJZ as an adjunct therapy for NSCLC patients

Apoptosis · Biology · Decoction · Downregulation and upregulation · EZH2 · Gene · Inhibitory postsynaptic potential · Methylation · PI3K/AKT/mTOR pathway · Protein kinase B · Traditional medicine · Cancer-related molecular mechanisms research · Chemistry · Immune Cell Function and Interaction · Medicine · T-cell and B-cell Immunology · Biochemistry · Cancer Research · Internal Medicine · Pharmacology

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    Open Access•Francesca Bray, Mathieu Laversanne et al.•CA: A Cancer Journal for Clinicians•2024

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