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Evidence for increased DNA damage repair in the postmortem brain of the high stress-response group of schizophrenia

Datos Bibliográficos

ID15519580
AutoresRisa Shishido (Fukushima Medical University), Yasuto Kunii (0000-0003-1569-7819, Fukushima Medical University, autor de correspondencia), Mizuki Hino (0000-0003-3700-0322, Fukushima Medical University), Ryuta Izumi (0000-0002-8453-3474, Fukushima Medical University), Atsuko Nagaoka (0000-0002-1159-4436, Fukushima Medical University), Hideki Hayashi (0000-0002-4693-9801, Niigata University), Akiyoshi Kakita (0000-0003-4244-0277, Niigata University), Hiroaki Tomita (0000-0003-2628-880X, Tohoku University), Hirooki Yabe (0000-0003-2668-129X, Fukushima Medical University)
Año2023
Volumen14
Páginas1183696-1183696
Fecha de publicación2023-08-22
Peer ReviewedSí
Open AccessSí
TipoARTICLE
RevistaFrontiers in Psychiatry (JOURNAL)
Identificadores de la revistaISSN: 1664-0640 • E-ISSN: 1664-0640
EditorialFrontiers Media (PUBLISHER • CH)
DOI10.3389/fpsyt.2023.1183696
PMID37674553
OpenAlexW4386070413
IdiomaEN
Referencias citadas59

Our results suggest that there are subgroups of SZ with different degrees of stress impact. Furthermore, the pathophysiology of these subgroups may be associated with DNA damage repair. These results provide new insights into the interactions and heterogeneity between genetic and environmental factors

Biology · DNA damage · Glucocorticoid receptor · Population · Prefrontal cortex · Receptor · Medicine · Neuroinflammation and Neurodegeneration Mechanisms · Neuroscience · Stress Responses and Cortisol · Tryptophan and brain disorders · Endocrinology · Genetics · Internal Medicine

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