Matthew R Hayes
Biographic Data
| ID | 10899089 |
|---|---|
| NAME | Matthew R Hayes |
| GIVEN NAMES | Matthew R |
| FAMILY NAME | Hayes |
| SIGNATURE | HAYES M R |
| AFFILIATIONS | University of Pennsylvania |
| ORCID | 0000-0001-9782-6551 |
| VERIFIED | No |
| TOTAL WORKS | 12 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 12 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2006 |
| LATEST PUBLICATION YEAR | 2011 |
| H-INDEX | 0 |
Central and vagal mechanisms of glucagon-like-peptide-1 receptor-mediated suppression of food intake
Pomc-PTP1B−/− mice show increased sensitivity to hindbrain MTII-induced reductions of food intake
NTS GLP-1 projections to the mesolimbic dopaminergic reward system reduce food intake
Glucagon-like peptide-1 receptor agonists suppress water intake
NTS leptin signaling contributes to meal size control and suppression of food intake by intestinally-derived satiation signals
Hindbrain BDNF activity reduces food intake through modulation of energy status signaling
Hindbrain leptin and GLP-1 receptor signaling interact in food intake control
Hippocampal leptin signaling reduces food intake and body weight and influences appetitive spatial memory
Endogenous leptin signaling in the NTS is required for energy balance regulation
Contribution of hindbrain GLP-1 receptor activation to feeding control—Role of endogenous GLP-1 and of ERK/amp-kinase pathways
The caudal brainstem is sufficient to mediate inhibition of gastric emptying by peripheral glucagon-like-peptide-1 receptor activation
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The caudal brainstem is sufficient to mediate inhibition of gastric emptying by peripheral glucagon-like-peptide-1 receptor activation
Contribution of hindbrain GLP-1 receptor activation to feeding control—Role of endogenous GLP-1 and of ERK/amp-kinase pathways
Endogenous leptin signaling in the NTS is required for energy balance regulation
Hindbrain BDNF activity reduces food intake through modulation of energy status signaling
Hindbrain leptin and GLP-1 receptor signaling interact in food intake control
Hippocampal leptin signaling reduces food intake and body weight and influences appetitive spatial memory
Central and vagal mechanisms of glucagon-like-peptide-1 receptor-mediated suppression of food intake
Pomc-PTP1B−/− mice show increased sensitivity to hindbrain MTII-induced reductions of food intake
NTS GLP-1 projections to the mesolimbic dopaminergic reward system reduce food intake
Glucagon-like peptide-1 receptor agonists suppress water intake
NTS leptin signaling contributes to meal size control and suppression of food intake by intestinally-derived satiation signals
Medicine (12 works) · Endocrinology (11 works) · Internal Medicine (11 works) · Regulation of Appetite and Obesity (8 works) · Food intake (7 works) · Central nervous system (6 works) · Receptor (6 works) · Hindbrain (5 works) · Neuroscience (5 works) · Agonist (4 works)