Gema Medina‐Gómez
Biographic Data
| ID | 10938265 |
|---|---|
| NAME | Gema Medina‐Gómez |
| GIVEN NAMES | Gema |
| FAMILY NAME | Medina‐Gómez |
| SIGNATURE | GOMEZ G M |
| AFFILIATIONS | University of Cambridge |
| ORCID | 0000-0001-8169-681X |
| VERIFIED | No |
| TOTAL WORKS | 1 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 1 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2007 |
| LATEST PUBLICATION YEAR | 2007 |
| H-INDEX | 0 |
Adipogenesis and lipotoxicity
Obesity is characterised by an increase in the adipose deposits, resulting from an imbalance between food intake and energy expenditure. When expansion of the adipose tissue reaches its maximum limit, as in obesity, fat accumulates in non-adipose tissues such as liver, heart, muscle and pancreas, developing a toxic response known as lipotoxicity, a condition that promotes the development of insulin resistance and other metabolic complications. Th…
No prominent works on this page.
Adipogenesis and lipotoxicity
Obesity is characterised by an increase in the adipose deposits, resulting from an imbalance between food intake and energy expenditure. When expansion of the adipose tissue reaches its maximum limit, as in obesity, fat accumulates in non-adipose tissues such as liver, heart, muscle and pancreas, developing a toxic response known as lipotoxicity, a condition that promotes the development of insulin resistance and other metabolic complications. Th…
Adipogenesis (1 work) · Adipokines, Inflammation, and Metabolic Diseases (1 work) · Adipose tissue (1 work) · Adipose Tissue and Metabolism (1 work) · Biochemistry (1 work) · Biology (1 work) · Coactivator (1 work) · Endocrinology (1 work) · Insulin (1 work) · Insulin resistance (1 work)