Brian F Hogan
Biographic Data
| ID | 2497599 |
|---|---|
| NAME | Brian F Hogan |
| GIVEN NAMES | Brian F |
| FAMILY NAME | Hogan |
| SIGNATURE | HOGAN B F |
| AFFILIATIONS | University of Toronto |
| VERIFIED | No |
| TOTAL WORKS | 3 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 3 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 1981 |
| LATEST PUBLICATION YEAR | 2006 |
| H-INDEX | 0 |
Arrestin-2 and G Protein-coupled Receptor Kinase 5 Interact with NFκB1 p105 and Negatively Regulate Lipopolysaccharide-stimulated ERK1/2 Activation in Macrophages
Toll-like receptors (TLRs) are a recently described receptor class involved in the regulation of innate and adaptive immunity. Here, we demonstrate that arrestin-2 and GRK5 (G protein-coupled receptor kinase 5), proteins that regulate G protein-coupled receptor signaling, play a negative role in TLR4 signaling in Raw264.7 macrophages. We find that lipopolysaccharide (LPS)-induced ERK1/2 phosphorylation is significantly enhanced in arrestin-2 and …
Church and Union
The Working Class in Northern Ontario
No prominent works on this page.
Church and Union
The Working Class in Northern Ontario
Arrestin-2 and G Protein-coupled Receptor Kinase 5 Interact with NFκB1 p105 and Negatively Regulate Lipopolysaccharide-stimulated ERK1/2 Activation in Macrophages
Toll-like receptors (TLRs) are a recently described receptor class involved in the regulation of innate and adaptive immunity. Here, we demonstrate that arrestin-2 and GRK5 (G protein-coupled receptor kinase 5), proteins that regulate G protein-coupled receptor signaling, play a negative role in TLR4 signaling in Raw264.7 macrophages. We find that lipopolysaccharide (LPS)-induced ERK1/2 phosphorylation is significantly enhanced in arrestin-2 and …
Political science (3 works) · Canadian Identity and History (2 works) · History (2 works) · Sociology (2 works) · Art (1 work) · Art history (1 work) · Artificial Intelligence (1 work) · Association (psychology (1 work) · Class (philosophy (1 work) · Computer Science (1 work)