Duk-Hee Lee
Biographic Data
| ID | 5469741 |
|---|---|
| NAME | Duk-Hee Lee |
| GIVEN NAMES | Duk-Hee |
| FAMILY NAME | Lee |
| SIGNATURE | LEE D |
| AFFILIATIONS | Kyungpook National University |
| ORCID | 0009-0000-3492-1199 |
| VERIFIED | Yes |
| TOTAL WORKS | 5 |
| TOTAL CITATIONS | 11 |
| WORKS AS AUTHOR | 5 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2006 |
| LATEST PUBLICATION YEAR | 2019 |
| H-INDEX | 2 |
New approaches to cope with possible harms of low-dose environmental chemicals
Low-dose environmental chemicals including endocrine-disrupting chemicals can disturb endocrine, nervous and immune systems. Traditional chemical-focused approaches, strict regulation and avoidance of exposure sources, can help protect humans from individual or several chemicals in the high-dose range, but their value in the low-dose range is questionable. First, exposure sources to problematic environmental chemicals are omnipresent, and many co…
Hormesis and public health
Exposure to persistent organic pollutants (POPs) is linked to many chronic diseases, including diabetes and cardiovascular diseases. Among several possible mechanisms are gradual glutathione depletion and mitochondrial dysfunction after chronic exposure to very low doses of POP mixtures. However, it is biologically noteworthy that glutathione status and mitochondrial function is subject to hormesis, defined broadly as mild stress-induced stimulat…
What is the role of human contamination by environmental chemicals in the development of type 1 diabetes
The increasing incidence of type 1 diabetes (T1D) in children around the world is unexplained. Even though various environmental chemicals have been linked to the development of type 2 diabetes as well as other autoimmune diseases, the possibility that environmental chemicals may contribute to the development of T1D has not been adequately evaluated. There is preliminary epidemiological evidence that exposure to certain chemicals, such as N-nitro…
Association of serum concentrations of persistent organic pollutants with the prevalence of learning disability and attention deficit disorder
Objective: Even though persistent organic pollutants (POPs) are well-known neurotoxicants, there is no previous study, even cross-sectional, on the association between background exposure to POPs and clinically significant developmental disorders, such as learning disability (LD) or attention deficit disorder (ADD), among children from a general population. Design: Cross-sectional study. Setting: Study subjects were 278 children aged 12–15 years …
Could low-level background exposure to persistent organic pollutants contribute to the social burden of type 2 diabetes
Persistent organic pollutants may contribute to cause diabetes
Association of serum concentrations of persistent organic pollutants with the prevalence of learning disability and attention deficit disorder
Objective: Even though persistent organic pollutants (POPs) are well-known neurotoxicants, there is no previous study, even cross-sectional, on the association between background exposure to POPs and clinically significant developmental disorders, such as learning disability (LD) or attention deficit disorder (ADD), among children from a general population. Design: Cross-sectional study. Setting: Study subjects were 278 children aged 12–15 years …
New approaches to cope with possible harms of low-dose environmental chemicals
Low-dose environmental chemicals including endocrine-disrupting chemicals can disturb endocrine, nervous and immune systems. Traditional chemical-focused approaches, strict regulation and avoidance of exposure sources, can help protect humans from individual or several chemicals in the high-dose range, but their value in the low-dose range is questionable. First, exposure sources to problematic environmental chemicals are omnipresent, and many co…
What is the role of human contamination by environmental chemicals in the development of type 1 diabetes
The increasing incidence of type 1 diabetes (T1D) in children around the world is unexplained. Even though various environmental chemicals have been linked to the development of type 2 diabetes as well as other autoimmune diseases, the possibility that environmental chemicals may contribute to the development of T1D has not been adequately evaluated. There is preliminary epidemiological evidence that exposure to certain chemicals, such as N-nitro…
Could low-level background exposure to persistent organic pollutants contribute to the social burden of type 2 diabetes
Persistent organic pollutants may contribute to cause diabetes
Hormesis and public health
Exposure to persistent organic pollutants (POPs) is linked to many chronic diseases, including diabetes and cardiovascular diseases. Among several possible mechanisms are gradual glutathione depletion and mitochondrial dysfunction after chronic exposure to very low doses of POP mixtures. However, it is biologically noteworthy that glutathione status and mitochondrial function is subject to hormesis, defined broadly as mild stress-induced stimulat…
Could low-level background exposure to persistent organic pollutants contribute to the social burden of type 2 diabetes
Persistent organic pollutants may contribute to cause diabetes
Association of serum concentrations of persistent organic pollutants with the prevalence of learning disability and attention deficit disorder
Objective: Even though persistent organic pollutants (POPs) are well-known neurotoxicants, there is no previous study, even cross-sectional, on the association between background exposure to POPs and clinically significant developmental disorders, such as learning disability (LD) or attention deficit disorder (ADD), among children from a general population. Design: Cross-sectional study. Setting: Study subjects were 278 children aged 12–15 years …
What is the role of human contamination by environmental chemicals in the development of type 1 diabetes
The increasing incidence of type 1 diabetes (T1D) in children around the world is unexplained. Even though various environmental chemicals have been linked to the development of type 2 diabetes as well as other autoimmune diseases, the possibility that environmental chemicals may contribute to the development of T1D has not been adequately evaluated. There is preliminary epidemiological evidence that exposure to certain chemicals, such as N-nitro…
Hormesis and public health
Exposure to persistent organic pollutants (POPs) is linked to many chronic diseases, including diabetes and cardiovascular diseases. Among several possible mechanisms are gradual glutathione depletion and mitochondrial dysfunction after chronic exposure to very low doses of POP mixtures. However, it is biologically noteworthy that glutathione status and mitochondrial function is subject to hormesis, defined broadly as mild stress-induced stimulat…
New approaches to cope with possible harms of low-dose environmental chemicals
Low-dose environmental chemicals including endocrine-disrupting chemicals can disturb endocrine, nervous and immune systems. Traditional chemical-focused approaches, strict regulation and avoidance of exposure sources, can help protect humans from individual or several chemicals in the high-dose range, but their value in the low-dose range is questionable. First, exposure sources to problematic environmental chemicals are omnipresent, and many co…
Environmental health (4 works) · Medicine (4 works) · Pollutant (3 works) · Biology (2 works) · Carcinogens and Genotoxicity Assessment (2 works) · Diabetes mellitus (2 works) · Effects and risks of endocrine disrupting chemicals (2 works) · Endocrinology (2 works) · Endocrinology (2 works) · Toxic Organic Pollutants Impact (2 works)