Thomas A Lutz
Biographic Data
| ID | 6257937 |
|---|---|
| NAME | Thomas A Lutz |
| GIVEN NAMES | Thomas A |
| FAMILY NAME | Lutz |
| SIGNATURE | LUTZ T A |
| AFFILIATIONS | University of Zurich |
| ORCID | 0000-0002-5056-8548 |
| VERIFIED | Yes |
| TOTAL WORKS | 34 |
| TOTAL CITATIONS | 6 |
| WORKS AS AUTHOR | 34 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2000 |
| LATEST PUBLICATION YEAR | 2022 |
| H-INDEX | 2 |
Creating the amylin story
This paper is based on a presentation given at the Annual Meeting of the Society for the Study of Ingestive Behavior in July 2021 and provides a personal view on some of the milestones in the discovery of amylin as a constituent of pancreatic islet amyloid deposits, as a pancreatic beta-cell hormone, and on its role in physiology and pathophysiology. Only selected effects of amylin are discussed here because we recently published extensive review…
Introduction to the special issue “bariatric surgery and appetite”
Introduction to the special issue “Bariatric Surgery and Appetite
La Shoah dans la culture mémorielle allemande
Rygb increases the satiating effect of intrajejunal lipid infusions in female rats
Amylin and the area postrema, but not only there
Rygb progressively increases avidity for a low-energy, artificially sweetened diet in female rats
Neural mechanisms of the eating-inhibitory effect of circulating glucagon-like peptide-1 (GLP-1) in rats
LPS inhibits ghrelin-excited neurons and food intake via central nitric oxide signaling
Amylin induced ERK 1/2 phosphorylation may contribute to its eating inhibitory effect
Involvement of the histaminergic system in amylin and leptin action
Estradiol (E2) increases the acute eating-inhibitory effect of amylin in ovariectomized (OVX) rats
The sensitivity of AP neurons to amylin and GLP-1 is modulated by the feeding status
Roux-en-Y gastric bypass in rats increases sucrose licking depending on deprivation state and trial taking in brief access tests
Short-term, but not extended, access to palatable diet diminishes amylin responsiveness in rat
Amylin induces ERK 1/2 phosphorylation in structures of the AP/NTS-LPB-Ce-BSTL axis
Nitric oxide contributes to LPS-induced anorexia
Diet-induced obesity, hyperamylinemia and amylin sensitivity
Roux-en-Y gastric bypass reduces bone mineral density independent of body weight in rats
Knock-down of estrogen receptor-α (ERα) neurons in the nucleus tractus solitarii (NTS) eliminates CCK-induced c-Fos expression in the paraventricular nucleus of the hypothalamus (PVN)
Selective RNAi knock-down of estrogen receptor-α (ER-α) neurons in the nucleus tractus solitarii (NTS) eliminates estradiol's (E2) inhibitory effect on food intake in ovariectomized (OVX) rats
Subacute endotoxemia temporarily impairs insulin sensitivity and beta-cell function in cats
Noradrenergic neurons of the area postrema mediate amylin's anorectic action
Amylin deficient mice have decreased fiber density in AP-NTS projections
The ability of amylin to reduce eating depends on the protein content of the diet
Creating the amylin story
This paper is based on a presentation given at the Annual Meeting of the Society for the Study of Ingestive Behavior in July 2021 and provides a personal view on some of the milestones in the discovery of amylin as a constituent of pancreatic islet amyloid deposits, as a pancreatic beta-cell hormone, and on its role in physiology and pathophysiology. Only selected effects of amylin are discussed here because we recently published extensive review…
Rygb progressively increases avidity for a low-energy, artificially sweetened diet in female rats
Rygb increases the satiating effect of intrajejunal lipid infusions in female rats
Transient hypophagia in rats switched from high-fat diets with different fatty-acid pattern to a high-carbohydrate diet
Withdrawn
Withdrawn
Withdrawn
Nutrients modulate amylin's effect on c-Fos expression in the area postrema and on food intake
Central modulation of the feeding response to amylin by leptin and insulin
Chronic central infusion of amylin lowers body weight gain independent of initial body weight in chow fed rats but not in rats after 3 weeks of Ensure® feeding
Effect of peripheral and central amylin on energy expenditure and body temperature
The central signaling pathways of amylin
Selective RNAi knock-down of estrogen receptor-α (ER-α) neurons in the nucleus tractus solitarii (NTS) eliminates estradiol's (E2) inhibitory effect on food intake in ovariectomized (OVX) rats
Subacute endotoxemia temporarily impairs insulin sensitivity and beta-cell function in cats
Noradrenergic neurons of the area postrema mediate amylin's anorectic action
Amylin deficient mice have decreased fiber density in AP-NTS projections
The ability of amylin to reduce eating depends on the protein content of the diet
Roux-en-Y gastric bypass in rats increases sucrose licking depending on deprivation state and trial taking in brief access tests
Short-term, but not extended, access to palatable diet diminishes amylin responsiveness in rat
Amylin induces ERK 1/2 phosphorylation in structures of the AP/NTS-LPB-Ce-BSTL axis
Nitric oxide contributes to LPS-induced anorexia
Diet-induced obesity, hyperamylinemia and amylin sensitivity
Roux-en-Y gastric bypass reduces bone mineral density independent of body weight in rats
Knock-down of estrogen receptor-α (ERα) neurons in the nucleus tractus solitarii (NTS) eliminates CCK-induced c-Fos expression in the paraventricular nucleus of the hypothalamus (PVN)
Neural mechanisms of the eating-inhibitory effect of circulating glucagon-like peptide-1 (GLP-1) in rats
LPS inhibits ghrelin-excited neurons and food intake via central nitric oxide signaling
Amylin induced ERK 1/2 phosphorylation may contribute to its eating inhibitory effect
Involvement of the histaminergic system in amylin and leptin action
Medicine (30 works) · Internal Medicine (28 works) · Endocrinology (27 works) · Amylin (18 works) · Chemistry (14 works) · Biology (12 works) · Psychology (12 works) · Neuroscience (11 works) · Regulation of Appetite and Obesity (11 works) · Diet and metabolism studies (10 works)