Sirong Zhang
Biographic Data
| ID | 7263405 |
|---|---|
| NAME | Sirong Zhang |
| GIVEN NAMES | Sirong |
| FAMILY NAME | Zhang |
| SIGNATURE | ZHANG S |
| AFFILIATIONS | Xinjiang Medical University |
| ORCID | 0009-0000-9582-8908 |
| VERIFIED | Yes |
| TOTAL WORKS | 2 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 2 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2024 |
| LATEST PUBLICATION YEAR | 2026 |
| H-INDEX | 0 |
Dual targeting by Artemisia rupestris L. disrupting TLR4/NF-κB signaling and macrophage driven immune evasion in hepatocellular carcinoma
ARL suppresses HCC progression by dual mechanisms that include the interference in TLR4/MyD88/NF-κB cascade to reduce proinflammatory cytokine production, and reprogramming macrophage polarization to attenuate immune driven tumor growth. These findings make ARL a promising natural therapeutic agent and provide an experimental basis for further in-depth subsequent exploration of the application of ARL in hepatocellular carcinoma
Exploring the molecular mechanism of Artemisia rupestris L. for the treatment of hepatocellular carcinoma via PI3K/AKT pathway
No prominent works on this page.
Exploring the molecular mechanism of Artemisia rupestris L. for the treatment of hepatocellular carcinoma via PI3K/AKT pathway
Dual targeting by Artemisia rupestris L. disrupting TLR4/NF-κB signaling and macrophage driven immune evasion in hepatocellular carcinoma
ARL suppresses HCC progression by dual mechanisms that include the interference in TLR4/MyD88/NF-κB cascade to reduce proinflammatory cytokine production, and reprogramming macrophage polarization to attenuate immune driven tumor growth. These findings make ARL a promising natural therapeutic agent and provide an experimental basis for further in-depth subsequent exploration of the application of ARL in hepatocellular carcinoma
Cytokine Signaling Pathways and Interactions (2 works) · Hepatocellular carcinoma (2 works) · Apoptosis (1 work) · Biochemistry (1 work) · Biology (1 work) · Cancer Research (1 work) · Cell Biology (1 work) · Cell growth (1 work) · Chemistry (1 work) · Computational Drug Discovery Methods (1 work)