Jia Ke
Biographic Data
| ID | 7268199 |
|---|---|
| NAME | Jia Ke |
| GIVEN NAMES | Jia |
| FAMILY NAME | Ke |
| SIGNATURE | KE J |
| AFFILIATIONS | Yueyang Hospital |
| ORCID | 0000-0002-6072-9800 |
| VERIFIED | Yes |
| TOTAL WORKS | 2 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 2 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2024 |
| LATEST PUBLICATION YEAR | 2026 |
| H-INDEX | 0 |
Prophylactic isoimperatorin protects against LPS-ALI via mTOR-dependent control of inflammation, oxidative stress and ferroptosis markers
ISO protected against LPS-induced ALI under a prophylactic dosing protocol via mTOR-dependent signaling (predominantly mTORC1), thereby restraining inflammation, oxidative stress, and ferroptosis-related markers. These findings support ISO as a mechanistically informed prophylactic lead that warrants evaluation under post-insult therapeutic regimens
Huzhangqingmaiyin protected vascular endothelial cells against cerebral small vessel disease through inhibiting inflammation
HZQMY could regulate the levels of key targets such as IL-1β、ICAM-1、VCAM-1 and VEGF, so as to achieve the purpose of treating CSVD
No prominent works on this page.
Huzhangqingmaiyin protected vascular endothelial cells against cerebral small vessel disease through inhibiting inflammation
HZQMY could regulate the levels of key targets such as IL-1β、ICAM-1、VCAM-1 and VEGF, so as to achieve the purpose of treating CSVD
Prophylactic isoimperatorin protects against LPS-ALI via mTOR-dependent control of inflammation, oxidative stress and ferroptosis markers
ISO protected against LPS-induced ALI under a prophylactic dosing protocol via mTOR-dependent signaling (predominantly mTORC1), thereby restraining inflammation, oxidative stress, and ferroptosis-related markers. These findings support ISO as a mechanistically informed prophylactic lead that warrants evaluation under post-insult therapeutic regimens
Angiogenesis (1 work) · Apoptosis (1 work) · Biochemistry (1 work) · Biology (1 work) · Cancer Research (1 work) · Cell (1 work) · Cell Biology (1 work) · Dosing (1 work) · Endothelial stem cell (1 work) · Ferroptosis and cancer prognosis (1 work)