Junzheng Yang
Biographic Data
| ID | 7281945 |
|---|---|
| NAME | Junzheng Yang |
| GIVEN NAMES | Junzheng |
| FAMILY NAME | Yang |
| SIGNATURE | YANG J |
| AFFILIATIONS | Guangzhou University of Chinese Medicine |
| ORCID | 0000-0001-9992-3179 |
| VERIFIED | Yes |
| TOTAL WORKS | 4 |
| TOTAL CITATIONS | 0 |
| AUTHOR COUNT | 4 |
| EDITOR COUNT | 0 |
| FIRST PUBLICATION YEAR | 2021 |
| LATEST PUBLICATION YEAR | 2025 |
| H-INDEX | 0 |
Sanqi Qushi formula ameliorates renal injury in experimental membranous nephropathy rats by inhibiting the MEK/ERK signaling pathway
SQQS ameliorates MN progression by inhibiting the MEK/ERK pathway, suppressing EMT, and reducing macrophage recruitment, with methylnissolin-3-O-glucoside as a key bioactive component
Serum metabolomics and 16S rRNA amplicon sequencing reveal the role of puerarin in alleviating bone loss aggravated by antidiabetic agent pioglitazone in type 2 diabetic mice
Bioinformatics analysis combined with experimental validation to explore the mechanism of XianLing GuBao capsule against osteoarthritis
Sanhuang Jiangtang tablet protects type 2 diabetes osteoporosis via AKT-GSK3β-NFATc1 signaling pathway by integrating bioinformatics analysis and experimental validation
No prominent works on this page.
Sanhuang Jiangtang tablet protects type 2 diabetes osteoporosis via AKT-GSK3β-NFATc1 signaling pathway by integrating bioinformatics analysis and experimental validation
Bioinformatics analysis combined with experimental validation to explore the mechanism of XianLing GuBao capsule against osteoarthritis
Sanqi Qushi formula ameliorates renal injury in experimental membranous nephropathy rats by inhibiting the MEK/ERK signaling pathway
SQQS ameliorates MN progression by inhibiting the MEK/ERK pathway, suppressing EMT, and reducing macrophage recruitment, with methylnissolin-3-O-glucoside as a key bioactive component
Serum metabolomics and 16S rRNA amplicon sequencing reveal the role of puerarin in alleviating bone loss aggravated by antidiabetic agent pioglitazone in type 2 diabetic mice
Biology (4 works) · Medicine (4 works) · Diabetes mellitus (3 works) · Endocrinology (3 works) · Gene (3 works) · Pharmacology (3 works) · Biochemistry (2 works) · Bioinformatics (2 works) · Bone Metabolism and Diseases (2 works) · Gene expression (2 works)