Jiang Zheng
Biographic Data
| ID | 7290001 |
|---|---|
| NAME | Jiang Zheng |
| GIVEN NAMES | Jiang |
| FAMILY NAME | Zheng |
| SIGNATURE | ZHENG J |
| AFFILIATIONS | Shenyang Pharmaceutical University |
| ORCID | 0009-0000-6181-4110 |
| VERIFIED | Yes |
| TOTAL WORKS | 4 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 4 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2015 |
| LATEST PUBLICATION YEAR | 2026 |
| H-INDEX | 0 |
CYP2C19-mediated metabolic activation and OAT3/P-gp-associated transport contribute to Dioscorea bulbifera L.-induced hepatotoxicity
Elucidation of Cortex Dictamni hepatotoxicity mechanisms through toxic component identification and integrated multi-omics analysis
Association of iron status with visual field loss in US females
This study revealed a negative association between TS and the prevalence of VFL among females aged 40-49 in the United States. Furthermore, ID, defined by both ferritin and MCV models, is linked to an elevated risk of VFL. Nevertheless, due to the limitations inherent in cross-sectional design, establishing a causal relationship between iron status and VFL remains challenging
Comparative studies of pharmacokinetics and anticoagulatory effect in rats after oral administration of Frankincense and its processed products
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Comparative studies of pharmacokinetics and anticoagulatory effect in rats after oral administration of Frankincense and its processed products
Association of iron status with visual field loss in US females
This study revealed a negative association between TS and the prevalence of VFL among females aged 40-49 in the United States. Furthermore, ID, defined by both ferritin and MCV models, is linked to an elevated risk of VFL. Nevertheless, due to the limitations inherent in cross-sectional design, establishing a causal relationship between iron status and VFL remains challenging
CYP2C19-mediated metabolic activation and OAT3/P-gp-associated transport contribute to Dioscorea bulbifera L.-induced hepatotoxicity
Elucidation of Cortex Dictamni hepatotoxicity mechanisms through toxic component identification and integrated multi-omics analysis
Drug-Induced Hepatotoxicity and Protection (2 works) · Medicine (2 works) · Metabolic pathway (2 works) · Acute Ischemic Stroke Management (1 work) · Anemia (1 work) · Biochemistry (1 work) · Biological activity (1 work) · Biostatistics (1 work) · Bone Metabolism and Diseases (1 work) · Cerebral Venous Sinus Thrombosis (1 work)