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Peter Gass

Biographic Data

ID7372435
NAMEPeter Gass
GIVEN NAMESPeter
FAMILY NAMEGass
SIGNATUREGASS P
AFFILIATIONSCentral Institute of Mental Health
ORCID0000-0003-3959-6369
VERIFIEDYes
TOTAL WORKS3
TOTAL CITATIONS0
WORKS AS AUTHOR3
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2018
LATEST PUBLICATION YEAR2021
H-INDEX0
  • Cre-Activation in ErbB4-Positive Neurons of Floxed Grin1/NMDA Receptor Mice Is Not Associated With Major Behavioral Impairment

    Open Access•Anne Stephanie Mallien, Natascha Pfeiffer et al.•ARTICLE•Frontiers in Psychiatry•2021

    Extensive evidence suggests a dysfunction of the glutamate NMDA receptor (NMDAR) in schizophrenia, a severe psychiatric disorder with putative early neurodevelopmental origins, but clinical onset mainly during late adolescence. On the other hand, pharmacological models using NMDAR antagonists and the clinical manifestation of anti-NMDAR encephalitis indicate that NMDAR blockade/hypofunction can trigger psychosis also at adult stages, without any …

  • Two Sides of the Same Coin

    Open Access•Dimitri Hefter, Cristina E Topor et al.•ARTICLE•Frontiers in Psychiatry•2019

    Background: Catatonic phenomena such as stupor, mutism, stereotypy, echolalia, echopraxia, affective flattening, psychomotor deficits, and social withdrawal are characteristic symptoms of both schizophrenia and autism spectrum disorders (ASD), suggesting overlapping pathophysiological similarities such as altered glutamatergic and dopaminergic synaptic transmission and common genetic mutations. In daily clinical practice, ASD can be masked by man…

  • Perinatal Hypoxia and Ischemia in Animal Models of Schizophrenia

    Open Access•Dimitri Hefter, Hugo H Marti et al.•ARTICLE•Frontiers in Psychiatry•2018

    Intrauterine or perinatal complications constitute a major risk for psychiatric diseases. Infants who suffered from hypoxia-ischemia (HI) are at twofold risk to develop schizophrenia in later life. Several animal models attempt to reproduce these complications to study the yet unknown steps between an insult in early life and outbreak of the disease decades later. However, it is very challenging to find the right type and severity of insult leadi…

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  • Perinatal Hypoxia and Ischemia in Animal Models of Schizophrenia

    Open Access•Dimitri Hefter, Hugo H Marti et al.•ARTICLE•Frontiers in Psychiatry•2018

    Intrauterine or perinatal complications constitute a major risk for psychiatric diseases. Infants who suffered from hypoxia-ischemia (HI) are at twofold risk to develop schizophrenia in later life. Several animal models attempt to reproduce these complications to study the yet unknown steps between an insult in early life and outbreak of the disease decades later. However, it is very challenging to find the right type and severity of insult leadi…

  • Two Sides of the Same Coin

    Open Access•Dimitri Hefter, Cristina E Topor et al.•ARTICLE•Frontiers in Psychiatry•2019

    Background: Catatonic phenomena such as stupor, mutism, stereotypy, echolalia, echopraxia, affective flattening, psychomotor deficits, and social withdrawal are characteristic symptoms of both schizophrenia and autism spectrum disorders (ASD), suggesting overlapping pathophysiological similarities such as altered glutamatergic and dopaminergic synaptic transmission and common genetic mutations. In daily clinical practice, ASD can be masked by man…

  • Cre-Activation in ErbB4-Positive Neurons of Floxed Grin1/NMDA Receptor Mice Is Not Associated With Major Behavioral Impairment

    Open Access•Anne Stephanie Mallien, Natascha Pfeiffer et al.•ARTICLE•Frontiers in Psychiatry•2021

    Extensive evidence suggests a dysfunction of the glutamate NMDA receptor (NMDAR) in schizophrenia, a severe psychiatric disorder with putative early neurodevelopmental origins, but clinical onset mainly during late adolescence. On the other hand, pharmacological models using NMDAR antagonists and the clinical manifestation of anti-NMDAR encephalitis indicate that NMDAR blockade/hypofunction can trigger psychosis also at adult stages, without any …

Medicine (3 works) · Psychiatry (3 works) · Psychology (3 works) · Schizophrenia (object-oriented programming (3 works) · Neuroscience (2 works) · Anxiety (1 work) · Attention Deficit Hyperactivity Disorder (1 work) · Autism (1 work) · Bipolar Disorder and Treatment (1 work) · Catatonia (1 work)

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