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Birte Glenthøj

Biographic Data

ID7389477
NAMEBirte Glenthøj
GIVEN NAMESBirte
FAMILY NAMEGlenthøj
SIGNATUREGLENTHØJ B
AFFILIATIONSGlostrup Hospital
ORCID0000-0003-3056-7262
VERIFIEDYes
TOTAL WORKS6
TOTAL CITATIONS0
WORKS AS AUTHOR6
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2018
LATEST PUBLICATION YEAR2024
H-INDEX0
  • Negative symptoms and social cognition as mediators of the relationship between neurocognition and functional outcome in schizophrenia

    Open Access•Giulia Maria Giordano, Pasquale Pezzella et al.•ARTICLE•Frontiers in Psychiatry•2024

    Our results indicate that pathways to functional outcomes are specific for different domains of real-life functioning and that negative symptoms and social cognition mediate the impact of neurocognitive deficits on different domains of functioning. Our results suggest that both negative symptoms and social cognition should be targeted by psychosocial interventions to enhance the functional impact of neurocognitive remediation

  • European Validation of the Self-Evaluation of Negative Symptoms (SNS)

    Open Access•Sonia Dollfus, Armida Mucci et al.•ARTICLE•Frontiers in Psychiatry•2022

    In a large European multicentric sample, this study demonstrated that the SNS has: (1) good psychometric properties with good convergent and divergent validities; (2) a five-factor latent structure; (3) an association with patients' functional outcome; and (4) the capacity to identify subjects with negative symptoms that is close to the BNSS and superior to the PANSS negative subscale

  • Add-On MEmaNtine to Dopamine Antagonism to Improve Negative Symptoms at First Psychosis- the Amend Trial Protocol

    Open Access•Katharina Oravsky Sandström, Olga B Baltzersen et al.•ARTICLE•Frontiers in Psychiatry•2022

    [ClinicalTrials.gov], identifier [NCT04789915]

  • Differential Effects of Aripiprazole and Amisulpride on Negative and Cognitive Symptoms in Patients With First-Episode Psychoses

    Open Access•Mette Ødegaard Nielsen, Tina Dam Kristensen et al.•ARTICLE•Frontiers in Psychiatry•2022

    Patients treated with aripiprazole improved on negative symptoms, which was not the case for patients treated with amisulpride. This may point to a general effect of a partial D2 receptor agonist on negative symptoms in patients with first-episode psychoses. There was, however, no improvement in cognitive functions

  • No Effects of Cognitive Remediation on Cerebral White Matter in Individuals at Ultra-High Risk for Psychosis—A Randomized Clinical Trial

    Open Access•Tina Dam Kristensen, Bjørn H Ebdrup et al.•ARTICLE•Frontiers in Psychiatry•2020

    ClinicalTrials.gov, identifier NCT02098408

  • Glutamate Levels and Resting Cerebral Blood Flow in Anterior Cingulate Cortex Are Associated at Rest and Immediately Following Infusion of S-Ketamine in Healthy Volunteers

    Open Access•Kirsten Borup Bojesen, Kasper Aagaard Andersen et al.•ARTICLE•Frontiers in Psychiatry•2018

    Progressive loss of brain tissue is seen in some patients with schizophrenia and might be caused by increased levels of glutamate and resting cerebral blood flow (rCBF) alterations. Animal studies suggest that the normalisation of glutamate levels decreases rCBF and prevents structural changes in hippocampus. However, the relationship between glutamate and rCBF in anterior cingulate cortex (ACC) of humans has not been studied in the absence of an…

No prominent works on this page.

  • Glutamate Levels and Resting Cerebral Blood Flow in Anterior Cingulate Cortex Are Associated at Rest and Immediately Following Infusion of S-Ketamine in Healthy Volunteers

    Open Access•Kirsten Borup Bojesen, Kasper Aagaard Andersen et al.•ARTICLE•Frontiers in Psychiatry•2018

    Progressive loss of brain tissue is seen in some patients with schizophrenia and might be caused by increased levels of glutamate and resting cerebral blood flow (rCBF) alterations. Animal studies suggest that the normalisation of glutamate levels decreases rCBF and prevents structural changes in hippocampus. However, the relationship between glutamate and rCBF in anterior cingulate cortex (ACC) of humans has not been studied in the absence of an…

  • No Effects of Cognitive Remediation on Cerebral White Matter in Individuals at Ultra-High Risk for Psychosis—A Randomized Clinical Trial

    Open Access•Tina Dam Kristensen, Bjørn H Ebdrup et al.•ARTICLE•Frontiers in Psychiatry•2020

    ClinicalTrials.gov, identifier NCT02098408

  • European Validation of the Self-Evaluation of Negative Symptoms (SNS)

    Open Access•Sonia Dollfus, Armida Mucci et al.•ARTICLE•Frontiers in Psychiatry•2022

    In a large European multicentric sample, this study demonstrated that the SNS has: (1) good psychometric properties with good convergent and divergent validities; (2) a five-factor latent structure; (3) an association with patients' functional outcome; and (4) the capacity to identify subjects with negative symptoms that is close to the BNSS and superior to the PANSS negative subscale

  • Add-On MEmaNtine to Dopamine Antagonism to Improve Negative Symptoms at First Psychosis- the Amend Trial Protocol

    Open Access•Katharina Oravsky Sandström, Olga B Baltzersen et al.•ARTICLE•Frontiers in Psychiatry•2022

    [ClinicalTrials.gov], identifier [NCT04789915]

  • Differential Effects of Aripiprazole and Amisulpride on Negative and Cognitive Symptoms in Patients With First-Episode Psychoses

    Open Access•Mette Ødegaard Nielsen, Tina Dam Kristensen et al.•ARTICLE•Frontiers in Psychiatry•2022

    Patients treated with aripiprazole improved on negative symptoms, which was not the case for patients treated with amisulpride. This may point to a general effect of a partial D2 receptor agonist on negative symptoms in patients with first-episode psychoses. There was, however, no improvement in cognitive functions

  • Negative symptoms and social cognition as mediators of the relationship between neurocognition and functional outcome in schizophrenia

    Open Access•Giulia Maria Giordano, Pasquale Pezzella et al.•ARTICLE•Frontiers in Psychiatry•2024

    Our results indicate that pathways to functional outcomes are specific for different domains of real-life functioning and that negative symptoms and social cognition mediate the impact of neurocognitive deficits on different domains of functioning. Our results suggest that both negative symptoms and social cognition should be targeted by psychosocial interventions to enhance the functional impact of neurocognitive remediation

Medicine (6 works) · Psychology (6 works) · Cognition (4 works) · Internal Medicine (4 works) · Psychiatry (4 works) · Schizophrenia research and treatment (4 works) · Clinical Psychology (3 works) · Clinical Psychology (3 works) · Neuroscience (3 works) · Schizophrenia (object-oriented programming (3 works)

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