Justin M O'Sullivan
Biographic Data
| ID | 7404494 |
|---|---|
| NAME | Justin M O'Sullivan |
| GIVEN NAMES | Justin M |
| FAMILY NAME | O'Sullivan |
| SIGNATURE | SULLIVAN J M O |
| AFFILIATIONS | University of Auckland |
| ORCID | 0000-0003-2927-450X |
| VERIFIED | Yes |
| TOTAL WORKS | 1 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 1 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2020 |
| LATEST PUBLICATION YEAR | 2020 |
| H-INDEX | 0 |
GWAS SNPs Impact Shared Regulatory Pathways Amongst Multimorbid Psychiatric Disorders and Cognitive Functioning
Background: Epidemiological research has reported that attention-deficit hyperactivity disorder (ADHD), anxiety, bipolar disorder (BD), schizophrenia (SCZ), and unipolar depression (UD) are multimorbid conditions that are typically accompanied by cognitive advantages or deficits, suggesting that common biological mechanisms may underlie these phenotypes. Genome-wide association studies (GWAS) have identified single-nucleotide polymorphisms (SNPs)…
No prominent works on this page.
GWAS SNPs Impact Shared Regulatory Pathways Amongst Multimorbid Psychiatric Disorders and Cognitive Functioning
Background: Epidemiological research has reported that attention-deficit hyperactivity disorder (ADHD), anxiety, bipolar disorder (BD), schizophrenia (SCZ), and unipolar depression (UD) are multimorbid conditions that are typically accompanied by cognitive advantages or deficits, suggesting that common biological mechanisms may underlie these phenotypes. Genome-wide association studies (GWAS) have identified single-nucleotide polymorphisms (SNPs)…
Anxiety (1 work) · Attention Deficit Hyperactivity Disorder (1 work) · Bioinformatics (1 work) · Biology (1 work) · Bipolar disorder (1 work) · Bipolar Disorder and Treatment (1 work) · Candidate gene (1 work) · Cognition (1 work) · Expression quantitative trait loci (1 work) · Gene (1 work)