Karl Bechter
Biographic Data
| ID | 7643693 |
|---|---|
| NAME | Karl Bechter |
| GIVEN NAMES | Karl |
| FAMILY NAME | Bechter |
| SIGNATURE | BECHTER K |
| AFFILIATIONS | Universität Ulm |
| ORCID | 0000-0003-4371-2932 |
| VERIFIED | Yes |
| TOTAL WORKS | 5 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 5 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 1995 |
| LATEST PUBLICATION YEAR | 2019 |
| H-INDEX | 0 |
Encephalitis, Mild Encephalitis, Neuroprogression, or Encephalopathy—Not Merely a Question of Terminology
Background: Psychoneuroimmunology research has presented emerging evidence of the involvement of inflammatory and immune mechanisms in the pathogenesis of severe mental disorders. In this context, new terms with increasing clinical relevance have been proposed, challenging the existing terms, and requiring consensus definitions of the new ones. Method: From a perspective of longstanding personal involvement in clinical settings and research in ps…
Schizophrenia or Atypical Lupus Erythematosus with Predominant Psychiatric Manifestations over 25 Years
We observed a case over 25 years of relapsing-remitting schizophrenic spectrum disorder, varying regarding the main symptomatology between more depressive or more schizoaffective or rather typical schizophrenic syndrome. Diseased phases were repeatedly accompanied by minor skin lesions, which were initially classified as mixed tissue disorder. Psychotic phases were waxing-waning over years. During one later relapse, skin involvement was severe, c…
IL1R2, CCR2, and CXCR4 May Form Heteroreceptor Complexes with NMDAR and D2R
The mild neuroinflammation hypothesis of schizophrenia was introduced by Bechter in 2001. It has been hypothesized that a hypofunction of glutamatergic signaling via N -methyl-D-aspartate receptors (NMDARs) and hyperactivation of dopamine D2 receptors play a role in schizophrenia. The triplet puzzle theory states that sets of triplet amino acid homologies guide two different receptors toward each other and contributes to the formation of a recept…
Chronic Cervicogenic Tinnitus Rapidly Resolved by Intermittent Use of Cervical Collar
In a single case of chronic tinnitus, we found that treatment with CC rapidly led to full remission. Blood flow reduction in vertebral arteries was unrelated to tinnitus. However, tinnitus could be resumed by constrained head postures. Experimental tinnitus replication (by inclination) points to an underscored role of upper posterior cervical muscle groups, matching with animal experiments, also in concert with other triggers including psychologi…
Research strategies in 'slow' infections in psychiatry
The research processes in the elucidation of the causes of general paresis, the first slow infection in psychiatry, and of Kuru, the first slow virus infection in man, were considered. The errors and difficulties encountered may contribute to the formulation of research strategies for contemporary work on possible persistent infections with unknown viruses as a cause of psychiatric disorders. Clinical observation, bold hypotheses and method ologi…
No prominent works on this page.
Research strategies in 'slow' infections in psychiatry
The research processes in the elucidation of the causes of general paresis, the first slow infection in psychiatry, and of Kuru, the first slow virus infection in man, were considered. The errors and difficulties encountered may contribute to the formulation of research strategies for contemporary work on possible persistent infections with unknown viruses as a cause of psychiatric disorders. Clinical observation, bold hypotheses and method ologi…
Chronic Cervicogenic Tinnitus Rapidly Resolved by Intermittent Use of Cervical Collar
In a single case of chronic tinnitus, we found that treatment with CC rapidly led to full remission. Blood flow reduction in vertebral arteries was unrelated to tinnitus. However, tinnitus could be resumed by constrained head postures. Experimental tinnitus replication (by inclination) points to an underscored role of upper posterior cervical muscle groups, matching with animal experiments, also in concert with other triggers including psychologi…
Schizophrenia or Atypical Lupus Erythematosus with Predominant Psychiatric Manifestations over 25 Years
We observed a case over 25 years of relapsing-remitting schizophrenic spectrum disorder, varying regarding the main symptomatology between more depressive or more schizoaffective or rather typical schizophrenic syndrome. Diseased phases were repeatedly accompanied by minor skin lesions, which were initially classified as mixed tissue disorder. Psychotic phases were waxing-waning over years. During one later relapse, skin involvement was severe, c…
IL1R2, CCR2, and CXCR4 May Form Heteroreceptor Complexes with NMDAR and D2R
The mild neuroinflammation hypothesis of schizophrenia was introduced by Bechter in 2001. It has been hypothesized that a hypofunction of glutamatergic signaling via N -methyl-D-aspartate receptors (NMDARs) and hyperactivation of dopamine D2 receptors play a role in schizophrenia. The triplet puzzle theory states that sets of triplet amino acid homologies guide two different receptors toward each other and contributes to the formation of a recept…
Encephalitis, Mild Encephalitis, Neuroprogression, or Encephalopathy—Not Merely a Question of Terminology
Background: Psychoneuroimmunology research has presented emerging evidence of the involvement of inflammatory and immune mechanisms in the pathogenesis of severe mental disorders. In this context, new terms with increasing clinical relevance have been proposed, challenging the existing terms, and requiring consensus definitions of the new ones. Method: From a perspective of longstanding personal involvement in clinical settings and research in ps…
Medicine (4 works) · Psychiatry (4 works) · Psychology (4 works) · Infectious Encephalopathies and Encephalitis (2 works) · Schizophrenia (object-oriented programming (2 works) · Tryptophan and brain disorders (2 works) · Adenosine and Purinergic Signaling (1 work) · Audiology (1 work) · Autoantibody (1 work) · Autoimmune encephalitis (1 work)