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Brian V Broberg

Biographic Data

ID7672998
NAMEBrian V Broberg
GIVEN NAMESBrian V
FAMILY NAMEBroberg
SIGNATUREBROBERG B V
AFFILIATIONSGlostrup Hospital
ORCID0000-0002-1215-6649
VERIFIEDYes
TOTAL WORKS2
TOTAL CITATIONS0
WORKS AS AUTHOR2
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2018
LATEST PUBLICATION YEAR2019
H-INDEX0
  • Bone Status in Obese, Non-diabetic, Antipsychotic-Treated Patients, and Effects of the Glucagon-Like Peptide-1 Receptor Agonist Exenatide on Bone Turnover Markers and Bone Mineral Density

    Open Access•Robert Eriksson, Brian V Broberg et al.•ARTICLE•Frontiers in Psychiatry•2019

    Background: Low bone mineral density (BMD) may constitute an underestimated comorbidity in schizophrenia patients undergoing long-term antipsychotic treatment. Glucagon-like peptide 1 (GLP-1) receptor agonists are antidiabetic drugs, which may also affect bone turnover. Methods: In planned secondary analyses of a 3 months, double-blind, randomized, placebo-controlled trial ( n = 45), we explored effects of the GLP-1 receptor agonist exenatide 2 m…

  • Glutamate Levels and Resting Cerebral Blood Flow in Anterior Cingulate Cortex Are Associated at Rest and Immediately Following Infusion of S-Ketamine in Healthy Volunteers

    Open Access•Kirsten Borup Bojesen, Kasper Aagaard Andersen et al.•ARTICLE•Frontiers in Psychiatry•2018

    Progressive loss of brain tissue is seen in some patients with schizophrenia and might be caused by increased levels of glutamate and resting cerebral blood flow (rCBF) alterations. Animal studies suggest that the normalisation of glutamate levels decreases rCBF and prevents structural changes in hippocampus. However, the relationship between glutamate and rCBF in anterior cingulate cortex (ACC) of humans has not been studied in the absence of an…

No prominent works on this page.

  • Glutamate Levels and Resting Cerebral Blood Flow in Anterior Cingulate Cortex Are Associated at Rest and Immediately Following Infusion of S-Ketamine in Healthy Volunteers

    Open Access•Kirsten Borup Bojesen, Kasper Aagaard Andersen et al.•ARTICLE•Frontiers in Psychiatry•2018

    Progressive loss of brain tissue is seen in some patients with schizophrenia and might be caused by increased levels of glutamate and resting cerebral blood flow (rCBF) alterations. Animal studies suggest that the normalisation of glutamate levels decreases rCBF and prevents structural changes in hippocampus. However, the relationship between glutamate and rCBF in anterior cingulate cortex (ACC) of humans has not been studied in the absence of an…

  • Bone Status in Obese, Non-diabetic, Antipsychotic-Treated Patients, and Effects of the Glucagon-Like Peptide-1 Receptor Agonist Exenatide on Bone Turnover Markers and Bone Mineral Density

    Open Access•Robert Eriksson, Brian V Broberg et al.•ARTICLE•Frontiers in Psychiatry•2019

    Background: Low bone mineral density (BMD) may constitute an underestimated comorbidity in schizophrenia patients undergoing long-term antipsychotic treatment. Glucagon-like peptide 1 (GLP-1) receptor agonists are antidiabetic drugs, which may also affect bone turnover. Methods: In planned secondary analyses of a 3 months, double-blind, randomized, placebo-controlled trial ( n = 45), we explored effects of the GLP-1 receptor agonist exenatide 2 m…

Internal Medicine (2 works) · Medicine (2 works) · Agonist (1 work) · Anesthesia (1 work) · Anterior cingulate cortex (1 work) · Antipsychotic (1 work) · Biochemistry (1 work) · Bone density (1 work) · Bone mineral (1 work) · Bone remodeling (1 work)

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