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Norman Junge

Biographic Data

ID8253030
NAMENorman Junge
GIVEN NAMESNorman
FAMILY NAMEJunge
SIGNATUREJUNGE N
AFFILIATIONSMedizinische Hochschule Hannover
ORCID0000-0002-3099-2667
VERIFIEDYes
TOTAL WORKS4
TOTAL CITATIONS0
WORKS AS AUTHOR4
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2022
LATEST PUBLICATION YEAR2023
H-INDEX0
  • Mild Crigler–Najjar Syndrome with Progressive Liver Disease—A Multicenter Retrospective Cohort Study

    Open Access•Norman Junge, Hanna Hentschel et al.•ARTICLE•Children•2023

    Crigler-Najjar Syndrome (CNS) with residual activity of UDP-glucuronosyltransferase 1A1 ( UGT1A1 ) and no need for daily phototherapy is called mild Crigler-Najjar Syndrome. Most of these patients need medical treatment for enzyme induction (phenobarbital) to lower blood levels of unconjugated bilirubin (UCB). Apart from this, no long-term problems have been described so far. The phenotype of patients with the homozygous pathogenic variant c.115C…

  • Intra-Abdominal Hypertension and Compartment Syndrome after Pediatric Liver Transplantation

    Open Access•Norman Junge, Annika Artmann et al.•ARTICLE•Children•2022

    In pediatric liver transplantation (pLT), the risk for the manifestation and relevance of intra-abdominal hypertension (IAH) and abdominal compartment syndrome (ACS) is high. This observational study aimed to evaluate the incidence, relevance and risk factors for IAH and ACS by monitoring the intra-abdominal pressure (IAP), macro- and microcirculation (near-infrared spectroscopy (NIRS)), clinical and laboratory status and outcomes of 27 patients …

  • Ileal Bile Acid Transporter Inhibition Reduces Post-Transplant Diarrhea and Growth Failure in FIC1 Disease—A Case Report

    Open Access•Johanna Ohlendorf, Imeke Goldschmidt et al.•ARTICLE•Children•2022

    Familial intrahepatic cholestasis 1 (FIC1) disease is a genetic disorder characterized by hepatic and gastrointestinal disease due to ATP8B1 deficiency, often requiring liver transplantation (LT). Extrahepatic symptoms, such as diarrhea, malabsorption, and failure to thrive, do not improve and instead may be aggravated after LT. We describe a patient with FIC1 disease who underwent LT at 2 years, 8 months of age. After LT, the child developed sev…

  • Cold Ischemia Time and Graft Fibrosis Are Associated with Autoantibodies after Pediatric Liver Transplantation

    Open Access•Norman Junge, Angelo Di Giorgio et al.•ARTICLE•Children•2022

    The reported prevalence of autoantibodies (AAB) (ANA, SMA, LKM, SLA) after pediatric liver transplantation (pLTX) varies considerably from 26-75%, but their clinical impact on outcome is uncertain. We aimed to study the prevalence of AAB after pLTX, their association with donor-, transplant-, and recipient-characteristics, and their relation to outcome. In our multicenter retrospective study, we aimed to clarify conflicting results from earlier s…

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  • Intra-Abdominal Hypertension and Compartment Syndrome after Pediatric Liver Transplantation

    Open Access•Norman Junge, Annika Artmann et al.•ARTICLE•Children•2022

    In pediatric liver transplantation (pLT), the risk for the manifestation and relevance of intra-abdominal hypertension (IAH) and abdominal compartment syndrome (ACS) is high. This observational study aimed to evaluate the incidence, relevance and risk factors for IAH and ACS by monitoring the intra-abdominal pressure (IAP), macro- and microcirculation (near-infrared spectroscopy (NIRS)), clinical and laboratory status and outcomes of 27 patients …

  • Ileal Bile Acid Transporter Inhibition Reduces Post-Transplant Diarrhea and Growth Failure in FIC1 Disease—A Case Report

    Open Access•Johanna Ohlendorf, Imeke Goldschmidt et al.•ARTICLE•Children•2022

    Familial intrahepatic cholestasis 1 (FIC1) disease is a genetic disorder characterized by hepatic and gastrointestinal disease due to ATP8B1 deficiency, often requiring liver transplantation (LT). Extrahepatic symptoms, such as diarrhea, malabsorption, and failure to thrive, do not improve and instead may be aggravated after LT. We describe a patient with FIC1 disease who underwent LT at 2 years, 8 months of age. After LT, the child developed sev…

  • Cold Ischemia Time and Graft Fibrosis Are Associated with Autoantibodies after Pediatric Liver Transplantation

    Open Access•Norman Junge, Angelo Di Giorgio et al.•ARTICLE•Children•2022

    The reported prevalence of autoantibodies (AAB) (ANA, SMA, LKM, SLA) after pediatric liver transplantation (pLTX) varies considerably from 26-75%, but their clinical impact on outcome is uncertain. We aimed to study the prevalence of AAB after pLTX, their association with donor-, transplant-, and recipient-characteristics, and their relation to outcome. In our multicenter retrospective study, we aimed to clarify conflicting results from earlier s…

  • Mild Crigler–Najjar Syndrome with Progressive Liver Disease—A Multicenter Retrospective Cohort Study

    Open Access•Norman Junge, Hanna Hentschel et al.•ARTICLE•Children•2023

    Crigler-Najjar Syndrome (CNS) with residual activity of UDP-glucuronosyltransferase 1A1 ( UGT1A1 ) and no need for daily phototherapy is called mild Crigler-Najjar Syndrome. Most of these patients need medical treatment for enzyme induction (phenobarbital) to lower blood levels of unconjugated bilirubin (UCB). Apart from this, no long-term problems have been described so far. The phenotype of patients with the homozygous pathogenic variant c.115C…

Internal Medicine (4 works) · Medicine (4 works) · Gastroenterology (3 works) · Liver transplantation (3 works) · Transplantation (3 works) · Disease (2 works) · Liver disease (2 works) · Liver Disease and Transplantation (2 works) · Liver Diseases and Immunity (2 works) · Organ Transplantation Techniques and Outcomes (2 works)

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