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Juan D Rodríguez-Callejas

Biographic Data

ID9104112
NAMEJuan D Rodríguez-Callejas
GIVEN NAMESJuan D
FAMILY NAMERodríguez-Callejas
SIGNATURERODRÍGUEZ-CALLEJAS J D
ORCID0000-0002-7365-1408
VERIFIEDYes
TOTAL WORKS1
TOTAL CITATIONS0
WORKS AS AUTHOR1
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2019
LATEST PUBLICATION YEAR2019
H-INDEX0
  • Loss of ferritin‐positive microglia relates to increased iron, RNA oxidation, and dystrophic microglia in the brains of aged male marmosets

    Open Access•Juan de Dios Rodríguez‐Callejas, Juan D Rodríguez-Callejas et al.•ARTICLE•American Journal of Primatology•2019

    Microglia are cells that protect brain tissue from invading agents and toxic substances, first by releasing pro-inflammatory cytokines, and thereafter by clearing tissue by phagocytosis. Microglia express ferritin, a protein with ferroxidase activity capable of storing iron, a metal that accumulates in brain during aging. Increasing evidence suggests that ferritin plays an important role in inflammation. However, it is not known if ferritin/iron …

No prominent works on this page.

  • Loss of ferritin‐positive microglia relates to increased iron, RNA oxidation, and dystrophic microglia in the brains of aged male marmosets

    Open Access•Juan de Dios Rodríguez‐Callejas, Juan D Rodríguez-Callejas et al.•ARTICLE•American Journal of Primatology•2019

    Microglia are cells that protect brain tissue from invading agents and toxic substances, first by releasing pro-inflammatory cytokines, and thereafter by clearing tissue by phagocytosis. Microglia express ferritin, a protein with ferroxidase activity capable of storing iron, a metal that accumulates in brain during aging. Increasing evidence suggests that ferritin plays an important role in inflammation. However, it is not known if ferritin/iron …

Alzheimer's disease research and treatments (1 work) · Barrier Structure and Function Studies (1 work) · Biochemistry (1 work) · Biology (1 work) · Cell Biology (1 work) · Chemistry (1 work) · Endocrinology (1 work) · Ferritin (1 work) · Hippocampal formation (1 work) · Immunology (1 work)

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