Kenneth A Mundt

Dados Biográficos

ID9996685
NOMEKenneth A Mundt
PRENOMESKenneth A
SOBRENOMEMundt
ASSINATURAMUNDT K A
AFILIAÇÕESUniversity of Massachusetts Amherst
ORCID0000-0002-9700-8679
VERIFICADOSim
TOTAL DE OBRAS7
TOTAL DE CITAÇÕES0
TOTAL COMO AUTOR7
TOTAL COMO EDITOR0
PRIMEIRO ANO DE PUBLICAÇÃO2022
ANO MAIS RECENTE DE PUBLICAÇÃO2025
ÍNDICE H0

  • Systematic review of the epidemiological evidence of associations between quantified occupational exposure to respirable crystalline silica and the risk of silicosis and lung cancer

    Open AccessKenneth A Mundt, William J Thompson et al.ARTICLEFrontiers in Public Health2025

  • Risk characterization for silica-related silicosis and lung cancer in communities adjacent to sand and gravel extraction facilities

    Open AccessAnthony J Russell, Kenneth A Mundt et al.ARTICLEFrontiers in Public Health2025

    Introduction: Effectively managing and reducing occupational exposure to crystalline silica continues to be a critical priority for public health agencies. The relationship between workplace inhalation exposure to respirable crystalline silica (RCS) and the onset of silicosis is well known. The body of evidence has shaped the evolution and promulgation of specific standards for the assessment and control of workplace RCS exposures. However, ongoi…

  • Results of 15 years of extended follow-up of the German porcelain workers cohort study

    Open AccessThomas Birk, Thomas J Birk et al.ARTICLEFrontiers in Public Health2025

    Objectives To quantify, after extending follow-up 15 years, the relationship between occupational respirable crystalline silica (RCS) exposure and risk of silicosis diagnosis and lung cancer mortality in the German Porcelain Workers Cohort Study, and to estimate possible exposure thresholds for these. Methods Porcelain workers enrolled between January 1, 1985, and December 31, 1987, in a mandatory medical surveillance program including triennial …

  • Using threshold Cox models to estimate change points in exposure-response relationships in an occupational epidemiological study of respirable crystalline silica and silicosis risk

    Open AccessDiezhang Wu, David Bin‐Chia Wu et al.ARTICLEFrontiers in Public Health2025

    Introduction: In occupational epidemiology, accurately quantifying exposure-response relationships is crucial. Methods: We introduce a threshold Cox model that includes a change point term to identify the optimal threshold. To address bias associated with maximum likelihood estimation under monotone likelihood, we employ Firth's penalized likelihood approach. The methodology was validated using simulation studies that evaluated model performance …

  • Framing diagnostic error

    Open AccessMary M Hunter, Montana Kekaimalu Hunter et al.ARTICLEFrontiers in Public Health2024

    Diagnostic errors burden the United States healthcare system. Depending on how they are defined, between 40,000 and 4 million cases occur annually. Despite this striking statistic, and the potential benefits epidemiological approaches offer in identifying risk factors for sub-optimal diagnoses, diagnostic error remains an underprioritized epidemiolocal research topic. Magnifying the challenge are the array of forms and definitions of diagnostic e…

  • Systematic review of the scientific evidence of the pulmonary carcinogenicity of talc

    Open AccessHeather N Lynch, Daniel J Lauer et al.ARTICLEFrontiers in Public Health2022

    We conducted a systematic review to assess the potential pulmonary carcinogenicity of inhaled talc in humans. Our systematic review methods adhere to Preferred Reporting Items for Systematic Reviews and Meta-Analyses (PRISMA) guidelines and incorporated aspects from the US Institute of Medicine (IOM) and several United States (US) Environmental Protection Agency (EPA) frameworks for systematic reviews. A comprehensive literature search was conduc…

  • Carcinogenicity of Poorly Soluble Low Toxicity Particles

    Open AccessKenneth A Mundt, Annette B Santamaria et al.ARTICLEFrontiers in Public Health2022

    Inhaled particles that are poorly soluble or insoluble and of low toxicity (“poorly soluble low toxicity” or “PSLT” particles), can accumulate in the lung and at lung overload levels induce lung cancers in rats. The question of whether PSLT particles increase lung cancer risk in humans is complicated by large differences between rats and humans and the relatively large particle doses administered in animal studies even when compared with heavy hu…