Weilong Song
Dados Biográficos
| ID | 7269502 |
|---|---|
| NOME | Weilong Song |
| PRENOMES | Weilong |
| SOBRENOME | Song |
| ASSINATURA | SONG W |
| AFILIAÇÕES | Wenzhou Medical University |
| ORCID | 0000-0001-5840-2713 |
| VERIFICADO | Sim |
| TOTAL DE OBRAS | 7 |
| TOTAL DE CITAÇÕES | 0 |
| TOTAL COMO AUTOR | 7 |
| TOTAL COMO EDITOR | 0 |
| PRIMEIRO ANO DE PUBLICAÇÃO | 2025 |
| ANO MAIS RECENTE DE PUBLICAÇÃO | 2026 |
| ÍNDICE H | 0 |
Gentiopicroside activates Pink1-dependent mitophagy to inhibit ferroptosis and promote flap survival
Sesamin promotes the survival of skin flaps in rats by inhibiting ferroptosis through the Nrf2/SLC7A11/GPX4 signaling pathway
Shuxuetong injection promotes skin flap survival by inhibiting pyroptosis mediated by the TLR4/NF-κB/NLRP3 signaling pathway
Quercetin enhances skin flap survival via Sirt1-regulated mitophagy promotion and pyroptosis inhibition
Osthole promotes skin flap survival by inhibiting ferroptosis and alleviating pyroptosis
Tectorigenin promotes skin flap survival by downregulating the TLR4–NF-κB signaling pathway
Tetrahydroxystilbene-2-O-beta-D-glucopyranoside promotes skin flap survival by promoting mitophagy through the Pink1/Parkin pathway
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Tectorigenin promotes skin flap survival by downregulating the TLR4–NF-κB signaling pathway
Tetrahydroxystilbene-2-O-beta-D-glucopyranoside promotes skin flap survival by promoting mitophagy through the Pink1/Parkin pathway
Gentiopicroside activates Pink1-dependent mitophagy to inhibit ferroptosis and promote flap survival
Sesamin promotes the survival of skin flaps in rats by inhibiting ferroptosis through the Nrf2/SLC7A11/GPX4 signaling pathway
Shuxuetong injection promotes skin flap survival by inhibiting pyroptosis mediated by the TLR4/NF-κB/NLRP3 signaling pathway
Quercetin enhances skin flap survival via Sirt1-regulated mitophagy promotion and pyroptosis inhibition
Osthole promotes skin flap survival by inhibiting ferroptosis and alleviating pyroptosis
Apoptosis (5 obras) · Cell survival (3 obras) · Ferroptosis and cancer prognosis (3 obras) · Inflammasome and immune disorders (3 obras) · Mitophagy (3 obras) · Oxidative stress (3 obras) · Programmed cell death (3 obras) · Pyroptosis (3 obras) · Signal transduction (3 obras) · Autophagy (2 obras)