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Air Pollution, Weather Stress, and Blood Pressure

Bibliographic Data

ID11021462
AuthorsWilliam S Linn (William S. Linn and Henry Gong Jr are with the Environmental Health Service, Rancho Los Amigos National Rehabilitation Center, Downey, Calif, and the Department of Preventive Medicine, Keck School of Medicine, University of Southern California, Los Angeles. Henry Gong Jr is also with the Department of Medicine, Rancho Los Amigos National Rehabilitation Center, and the Department of Medicine, Keck School of Medicine.), Henry Gong (William S. Linn and Henry Gong Jr are with the Environmental Health Service, Rancho Los Amigos National Rehabilitation Center, Downey, Calif, and the Department of Preventive Medicine, Keck School of Medicine, University of Southern California, Los Angeles. Henry Gong Jr is also with the Department of Medicine, Rancho Los Amigos National Rehabilitation Center, and the Department of Medicine, Keck School of Medicine.)
Year2001
Volume91
Issue9
Pages1345-b-1346
Publication date2001-09-01
Open AccessNo
TypeARTICLE
VenueAmerican Journal of Public Health (JOURNAL)
Journal identifiersISSN: 0090-0036 • E-ISSN: 1541-0048
PublisherAmerican Public Health Association (PUBLISHER • US)
DOI10.2105/ajph.91.9.1345-b
PMID11527746
OpenAlexW2049265017
LanguageEN
Citations received2
References cited2

Ibald-Mulli et al. recently reported a potentially important association between particulate air pollution and increased blood pressure and stated that this had not been documented previously.1 In fact, our research group previously reported a positive longitudinal relation between blood pressure and ambient particulate pollution.2 Our study differed markedly from Ibald-Mulli's in scale, duration, location, atmospheric conditions, and subject characteristics, as shown in Table 1. ▶ Nevertheless, certain key results—regression slopes (b) for blood pressure vs particulate pollution concentration—in the 2 studies were similar for our panel with chronic obstructive pulmonary disease (COPD) and for Ibald-Mulli's subgroups with identified risk factors (high plasma viscosity or heart rate). Our modestly larger values might reflect our measurement of particles with aerodynamic diameter <10 μm (PM10) rather than total suspended particles (TSP). TABLE 1— Effects of Air Pollution on Health: Comparative Results in Ibald-Mulli et al.1 and Linn et al.2 As Ibald-Mulli et al. pointed out, weather stresses or gaseous pollutants may confound particle–health relationships. Although we could not study confounders in detail, we found that our subjects' blood pressure appeared to respond to PM10 as measured in the outdoor background urban air environment, even though they spent most of their time inside their homes, where particle mass and chemical composition were noticeably different. Again, this seems consistent with the findings of Ibald-Mulli et al., based on outdoor background pollution measurements. On the basis of this evidence, we recommend wider use of simple noninvasive cardiovascular measurements in studies of air pollution and weather stresses. Increased understanding of response mechanisms, and clearer identification of populations at risk, should result

Air pollution · Biology · Blood pressure · Environmental health · Geography · Meteorology · Stress (linguistics) · Air Quality and Health Impacts · Air Quality Monitoring and Forecasting · Climate Change and Health Impacts · Ecology · Environmental Science · Internal Medicine · Medicine

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Citations per year0.09
Citation span2004 - 2006 (3)
Highly citedNo

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