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Xinyang tablet alleviated cardiac dysfunction in a cardiac pressure overload model by regulating the receptor-interacting serum/three-protein kinase 3/FUN14 domain containing 1-mediated mitochondrial unfolded protein response and mitophagy

Datos Bibliográficos

ID14733552
AutoresXin Dong (0000-0001-9908-4838, Guangzhou University of Chinese Medicine), Hao-wen Zhuang (Guangzhou University of Chinese Medicine), Rui-jia Wen, Ruijia Wen (Guangzhou University of Chinese Medicine), Yu-sheng Huang, Yu‐Sheng Huang (0000-0002-1119-7836, Guangzhou University of Chinese Medicine), Bing-xue Liang, Bingxue Liang, Huan Li (0000-0003-0910-1433, Guangzhou University of Chinese Medicine), Shao-xiang Xian, Shaoxiang Xian (0000-0002-9266-313X, Guangzhou University of Chinese Medicine), Chun Li (0000-0001-7981-7184, Guangzhou University of Chinese Medicine, autor de correspondencia), Ling-jun Wang, Lingjun Wang (0000-0001-6214-5182, Guangzhou University of Chinese Medicine, autor de correspondencia), Jun-yan Wang (0000-0002-2048-2225), Junyan Wang (0009-0007-1047-3783, Guangzhou University of Chinese Medicine, autor de correspondencia)
Año2024
Volumen330
Páginas118152
Fecha de publicación2024-08-01
Peer ReviewedSí
Open AccessSí
TipoARTICLE
RevistaJournal of Ethnopharmacology (JOURNAL)
Identificadores de la revistaISSN: 0378-8741 • E-ISSN: 1872-7573
EditorialElsevier BV (PUBLISHER)
DOI10.1016/j.jep.2024.118152
PMID38614260
OpenAlexW4394796334
IdiomaEN
Referencias citadas56

Angiotensin II · Apoptosis · Autophagy · Biology · Endoplasmic reticulum · Mitochondrion · Mitophagy · Pressure overload · Receptor · Unfolded protein response · Alzheimer's disease research and treatments · Autophagy in Disease and Therapy · Chemistry · Mitochondrial Function and Pathology · Biochemistry · Cell Biology · Endocrinology · Molecular Biology

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