Taraxacum mongolicum total triterpenoids and taraxasterol ameliorate benign prostatic hyperplasia by inhibiting androgen levels, inflammatory responses, and epithelial-mesenchymal transition via the TGFβ1/Smad signalling pathway
Dados Bibliográficos
Androgen · Biology · Epithelial–mesenchymal transition · Hyperplasia · Mesenchymal stem cell · Metastasis · SMAD · Traditional medicine · Transforming growth factor · Triterpenoid · Medicine · Phytoestrogen effects and research · Plant Toxicity and Pharmacological Properties · Sesquiterpenes and Asteraceae Studies · Cancer Research · Cell Biology · Endocrinology · Internal Medicine
The water-soluble subfraction from Artemisia argyi alleviates LPS-induced inflammatory responses via multiple pathways and targets in vitro and in vivo
Future aspects of plant derived bioactive metabolites as therapeutics to combat benign prostatic hyperplasia
Deciphering anti–benign prostatic hyperplasia potential of liangwanoside II based on metabolite profile characterization combined with targeted network pharmacology
Diterpene glycosides from Fructus Rubi ameliorates benign prostatic hyperplasia in rats through the androgen and TGF-β/Smad signaling pathway
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