R J Rodgers
Biographic Data
| ID | 6109669 |
|---|---|
| NAME | R J Rodgers |
| GIVEN NAMES | R J |
| FAMILY NAME | Rodgers |
| SIGNATURE | RODGERS R J |
| AFFILIATIONS | University of Bradford |
| VERIFIED | No |
| TOTAL WORKS | 8 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 8 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 1976 |
| LATEST PUBLICATION YEAR | 1995 |
| H-INDEX | 0 |
Neuropharmacological aspects of adaptive pain inhibition in murine “victims” of aggression
This review outlines recent research on a subset of physiological responses in murine “victims” or aggression. In a typical resident-intruder paradigm, the detailed study of intruders has revealed that exposure to conspecific attack (and related stimuli) is associated with two forms of analgesia which appear to be integral components of the murine defensive repertoire. In response to intense/enduring attack, intruder mice display a profound, long…
Pharmacological and neural control of anti-predator defense in the rat
An ethoexperimental analysis of the defensive behaviors of laboratory and wild rats to predators can predict specific defense patterns in terms of features of the threatening stimulus and the situation. This analysis suggests that a variety of specific defenses, such as flight, movement arrest, and defensive threat and attack, are most characteristic of fear to a present, discrete, threat stimulus while risk assessment patterns coupled with the i…
Naloxone differentially alters parental aggression by female mice towards conspecific intruders of differing sex
Lactating mice respond differently to male and female conspecific intruders, displaying defensive attack towards the former and offensive attack towards the latter. Two studies are reported in which the effects of naloxone on this differential response pattern were assessed. In the first study, lactating residents were pretreated with saline or naloxone (0.5, 2.5 mg/kg) and consecutively confronted with intruders of differing sex. Results suggest…
Endorphins, stress, and fighting behavior in rats
Alterations in shock-induced fighting and locomotor activity following intracerebroventricular injection of hydrocortisone in the rat
Three experiments were conducted in an attempt to clarify the facilitatory influence of hydrocortisone on shock-induced fighting in rats. Results of the first experiment indicated a biphasic, dose-dependent action of intraventricularly-administered hydrocortisone. Low (25 μg) and intermediate (50 μg) doses both facilitated fighting whilst the high (100 μg) dose exerted a potent suppressant effect. Two control tests were performed to determine whe…
The medial amygdala
Two aspects of the amygdaloid complex (corticomedial and basolateral) were examined with reference to serotonergic inhibition of shock-induced aggression. Fighting was significantly depressed by serotonergic stimulation (5-HT, 10 μg bilateral) in the corticomedial amygdala while serotonergic blockade (methysergide, 5 μg bilateral) in this region increased levels of fighting. No consistent effects were obtained with serotonergic manipulation of th…
Amygdaloid function in the central cholinergic mediation of shock-induced aggression in the rat
A series of experiments was designed to examine the role of central cholinergic mechanisms in shock-induced aggression. Cholinergic blockade in the basolateral amygdala, ventral hippocampus, or dorsal hippocampus resulted in greatly reduced levels of fighting in response to footshock. However, while pain sensitivity remained unaltered in the amygdala group, both of the hippocampal groups exhibited decreased shock sensitivity. Further investigatio…
Shock-induced aggression and pain sensitivity in the rat
The possible role of amygdaloid catecholamines in the control of shock-induced aggression and pain sensitivity in the rat was investigated. Bilateral microinjections of chlorpromazine into the corticomedial amygdala resulted in decreased fighting and decreased sensitivity to the shock stimulus. Further analysis of this effect, using specific adrenergic antagonists, revealed that neither a- nor Padrenergic systems appeared to be responsible for th…
No prominent works on this page.
Amygdaloid function in the central cholinergic mediation of shock-induced aggression in the rat
A series of experiments was designed to examine the role of central cholinergic mechanisms in shock-induced aggression. Cholinergic blockade in the basolateral amygdala, ventral hippocampus, or dorsal hippocampus resulted in greatly reduced levels of fighting in response to footshock. However, while pain sensitivity remained unaltered in the amygdala group, both of the hippocampal groups exhibited decreased shock sensitivity. Further investigatio…
Shock-induced aggression and pain sensitivity in the rat
The possible role of amygdaloid catecholamines in the control of shock-induced aggression and pain sensitivity in the rat was investigated. Bilateral microinjections of chlorpromazine into the corticomedial amygdala resulted in decreased fighting and decreased sensitivity to the shock stimulus. Further analysis of this effect, using specific adrenergic antagonists, revealed that neither a- nor Padrenergic systems appeared to be responsible for th…
The medial amygdala
Two aspects of the amygdaloid complex (corticomedial and basolateral) were examined with reference to serotonergic inhibition of shock-induced aggression. Fighting was significantly depressed by serotonergic stimulation (5-HT, 10 μg bilateral) in the corticomedial amygdala while serotonergic blockade (methysergide, 5 μg bilateral) in this region increased levels of fighting. No consistent effects were obtained with serotonergic manipulation of th…
Alterations in shock-induced fighting and locomotor activity following intracerebroventricular injection of hydrocortisone in the rat
Three experiments were conducted in an attempt to clarify the facilitatory influence of hydrocortisone on shock-induced fighting in rats. Results of the first experiment indicated a biphasic, dose-dependent action of intraventricularly-administered hydrocortisone. Low (25 μg) and intermediate (50 μg) doses both facilitated fighting whilst the high (100 μg) dose exerted a potent suppressant effect. Two control tests were performed to determine whe…
Endorphins, stress, and fighting behavior in rats
Naloxone differentially alters parental aggression by female mice towards conspecific intruders of differing sex
Lactating mice respond differently to male and female conspecific intruders, displaying defensive attack towards the former and offensive attack towards the latter. Two studies are reported in which the effects of naloxone on this differential response pattern were assessed. In the first study, lactating residents were pretreated with saline or naloxone (0.5, 2.5 mg/kg) and consecutively confronted with intruders of differing sex. Results suggest…
Pharmacological and neural control of anti-predator defense in the rat
An ethoexperimental analysis of the defensive behaviors of laboratory and wild rats to predators can predict specific defense patterns in terms of features of the threatening stimulus and the situation. This analysis suggests that a variety of specific defenses, such as flight, movement arrest, and defensive threat and attack, are most characteristic of fear to a present, discrete, threat stimulus while risk assessment patterns coupled with the i…
Neuropharmacological aspects of adaptive pain inhibition in murine “victims” of aggression
This review outlines recent research on a subset of physiological responses in murine “victims” or aggression. In a typical resident-intruder paradigm, the detailed study of intruders has revealed that exposure to conspecific attack (and related stimuli) is associated with two forms of analgesia which appear to be integral components of the murine defensive repertoire. In response to intense/enduring attack, intruder mice display a profound, long…
Psychology (8 works) · Medicine (7 works) · Internal Medicine (6 works) · Neuroendocrine regulation and behavior (6 works) · Aggression (5 works) · Neuroscience (5 works) · Endocrinology (4 works) · Neuropeptides and Animal Physiology (4 works) · Psychiatry (4 works) · Shock (circulatory) (4 works)