Guoqi Zhu
Biographic Data
| ID | 7259737 |
|---|---|
| NAME | Guoqi Zhu |
| GIVEN NAMES | Guoqi |
| FAMILY NAME | Zhu |
| SIGNATURE | ZHU G |
| AFFILIATIONS | Anhui University of Traditional Chinese Medicine |
| ORCID | 0000-0001-7432-6079 |
| VERIFIED | Yes |
| TOTAL WORKS | 7 |
| TOTAL CITATIONS | 0 |
| AUTHOR COUNT | 7 |
| EDITOR COUNT | 0 |
| FIRST PUBLICATION YEAR | 2021 |
| LATEST PUBLICATION YEAR | 2026 |
| H-INDEX | 0 |
Reconsidering college students’ low-carbon behavior
Purpose This study aims to investigate the mechanism of the influence of paradoxical thinking (PT), which characterizes the ability of college students to balance and integrate the conflict between hedonic and normative goals, on their campus low-carbon behaviors. Design/methodology/approach The conceptual model of “PT − Paradoxical salience (climate change concern, CCC) − Paradoxical acceptance (support for low-carbon behavioral norms, SN) − par…
Ginsenoside Rg2 delays brain aging via inhibiting α-synuclein expression and promoting FoxO-Mediated neurogenesis in mice
The mechanism underlying of Zuoguiyin on liver and kidney in D-gal-induced subacute aging female rats
This study reveals that ZGY exerts positive effects on the liver and kidney in D-gal-induced subacute aging female rats. Its mechanisms are associated with the activation of the SIRT1-PPARγ pathway, which regulates oxidative stress, inflammation, and apoptotic pathways
Polysaccharides from Polygonatum cyrtonema Hua prevent post-traumatic stress disorder behaviors in mice
Novel mechanisms of Anshen Dingzhi prescription against PTSD
Our data indicate that DCC is a key target for the regulation of synaptic function and inflammatory response in the onset of PTSD, and ADP likely reduces DCC to prevent PTSD via modulating downstream Rac1-PAK1 pathway. This study provides a novel mechanism for the onset of PTSD and warrants the clinical application of ADP
Exploring the effect of Anshen Dingzhi prescription on hippocampal mitochondrial signals in single prolonged stress mouse model
Polygonatum sibiricum polysaccharide prevents depression-like behaviors by reducing oxidative stress, inflammation, and cellular and synaptic damage
No prominent works on this page.
Polygonatum sibiricum polysaccharide prevents depression-like behaviors by reducing oxidative stress, inflammation, and cellular and synaptic damage
Polysaccharides from Polygonatum cyrtonema Hua prevent post-traumatic stress disorder behaviors in mice
Novel mechanisms of Anshen Dingzhi prescription against PTSD
Our data indicate that DCC is a key target for the regulation of synaptic function and inflammatory response in the onset of PTSD, and ADP likely reduces DCC to prevent PTSD via modulating downstream Rac1-PAK1 pathway. This study provides a novel mechanism for the onset of PTSD and warrants the clinical application of ADP
Exploring the effect of Anshen Dingzhi prescription on hippocampal mitochondrial signals in single prolonged stress mouse model
The mechanism underlying of Zuoguiyin on liver and kidney in D-gal-induced subacute aging female rats
This study reveals that ZGY exerts positive effects on the liver and kidney in D-gal-induced subacute aging female rats. Its mechanisms are associated with the activation of the SIRT1-PPARγ pathway, which regulates oxidative stress, inflammation, and apoptotic pathways
Reconsidering college students’ low-carbon behavior
Purpose This study aims to investigate the mechanism of the influence of paradoxical thinking (PT), which characterizes the ability of college students to balance and integrate the conflict between hedonic and normative goals, on their campus low-carbon behaviors. Design/methodology/approach The conceptual model of “PT − Paradoxical salience (climate change concern, CCC) − Paradoxical acceptance (support for low-carbon behavioral norms, SN) − par…
Ginsenoside Rg2 delays brain aging via inhibiting α-synuclein expression and promoting FoxO-Mediated neurogenesis in mice
Medicine (5 works) · Biochemistry (4 works) · Internal Medicine (4 works) · Oxidative stress (4 works) · Pharmacology (4 works) · Biology (3 works) · Hippocampal formation (3 works) · Hippocampus (3 works) · Inflammation (3 works) · Receptor (3 works)