Jialei Chen
Biographic Data
| ID | 7273740 |
|---|---|
| NAME | Jialei Chen |
| GIVEN NAMES | Jialei |
| FAMILY NAME | Chen |
| SIGNATURE | CHEN J |
| AFFILIATIONS | Chongqing Medical University |
| ORCID | 0009-0003-9259-5936 |
| VERIFIED | Yes |
| TOTAL WORKS | 2 |
| TOTAL CITATIONS | 0 |
| AUTHOR COUNT | 2 |
| EDITOR COUNT | 0 |
| FIRST PUBLICATION YEAR | 2025 |
| LATEST PUBLICATION YEAR | 2025 |
| H-INDEX | 0 |
Lexical Diversity and Syntactic Complexity in AI-Translated Legislative Texts
This study investigates the performance of artificial intelligence (AI) in the translation of legislative texts, focusing on the quality of translations produced by ChatGPT 4o and DeepL Pro. By using TAALED and NeoSCA, we evaluated and compared a number of indices in lexical diversity and syntactic complexity of AI-generated and human translations of twenty Chinese legislative texts. We used JASP to calculate Bayes Factor and then compare the tra…
Neuroprotective role of Da Qin Jiu decoction in ischemic stroke: Mitochondrial rescue through PI3K/Akt-mediated UPRmt activation
DQJD exerts neuroprotective effects in IS by restoring mitochondrial function through UPR mt activation via the PI3K/Akt pathway. These findings support further exploration of DQJD as a therapeutic option for IS
No prominent works on this page.
Lexical Diversity and Syntactic Complexity in AI-Translated Legislative Texts
This study investigates the performance of artificial intelligence (AI) in the translation of legislative texts, focusing on the quality of translations produced by ChatGPT 4o and DeepL Pro. By using TAALED and NeoSCA, we evaluated and compared a number of indices in lexical diversity and syntactic complexity of AI-generated and human translations of twenty Chinese legislative texts. We used JASP to calculate Bayes Factor and then compare the tra…
Neuroprotective role of Da Qin Jiu decoction in ischemic stroke: Mitochondrial rescue through PI3K/Akt-mediated UPRmt activation
DQJD exerts neuroprotective effects in IS by restoring mitochondrial function through UPR mt activation via the PI3K/Akt pathway. These findings support further exploration of DQJD as a therapeutic option for IS
Apoptosis (1 works) · Artificial Intelligence (1 works) · Biochemistry (1 works) · Biology (1 works) · Cardiology (1 works) · Computer Science (1 works) · Decoction (1 works) · Ischemia (1 works) · Ischemic stroke (1 works) · Law (1 works)