Zhong-Yan Zhou
Biographic Data
| ID | 7280491 |
|---|---|
| NAME | Zhong-Yan Zhou |
| GIVEN NAMES | Zhong-Yan |
| FAMILY NAME | Zhou |
| SIGNATURE | ZHOU Z |
| AFFILIATIONS | Longhua Hospital Shanghai University of Traditional Chinese Medicine |
| ORCID | 0000-0002-2449-1380 |
| VERIFIED | Yes |
| TOTAL WORKS | 4 |
| TOTAL CITATIONS | 0 |
| WORKS AS AUTHOR | 4 |
| WORKS AS EDITOR | 0 |
| FIRST PUBLICATION YEAR | 2015 |
| LATEST PUBLICATION YEAR | 2022 |
| H-INDEX | 0 |
Tribulus terrestris L. extract ameliorates atherosclerosis by inhibition of vascular smooth muscle cell proliferation in ApoE−/− mice and A7r5 cells via suppression of Akt/MEK/ERK signaling
FSEE presents potent anti- AS and VSMC proliferation activities and the underlying mechanism is likely to the suppression of Akt/MEK/ERK signaling. The active components of FSEE are JL-6 and its potential active pharmaceutical ingredients Tigo and Heco. So, FSEE and its active compounds may be potential therapeutic drug candidates for AS
Spatholobi Caulis extracts promote angiogenesis in Huvecs in vitro and in zebrafish embryos in vivo via up-regulation of VEGFRs
Amelioration of acute myocardial infarction by saponins from flower buds of Panax notoginseng via pro-angiogenesis and anti-apoptosis
Schisantherin A protects against 6-Ohda-induced dopaminergic neuron damage in zebrafish and cytotoxicity in SH-SY5Y cells through the ROS/NO and AKT/GSK3β pathways
No prominent works on this page.
Schisantherin A protects against 6-Ohda-induced dopaminergic neuron damage in zebrafish and cytotoxicity in SH-SY5Y cells through the ROS/NO and AKT/GSK3β pathways
Amelioration of acute myocardial infarction by saponins from flower buds of Panax notoginseng via pro-angiogenesis and anti-apoptosis
Spatholobi Caulis extracts promote angiogenesis in Huvecs in vitro and in zebrafish embryos in vivo via up-regulation of VEGFRs
Tribulus terrestris L. extract ameliorates atherosclerosis by inhibition of vascular smooth muscle cell proliferation in ApoE−/− mice and A7r5 cells via suppression of Akt/MEK/ERK signaling
FSEE presents potent anti- AS and VSMC proliferation activities and the underlying mechanism is likely to the suppression of Akt/MEK/ERK signaling. The active components of FSEE are JL-6 and its potential active pharmaceutical ingredients Tigo and Heco. So, FSEE and its active compounds may be potential therapeutic drug candidates for AS
Biochemistry (4 works) · Biology (4 works) · Medicine (4 works) · Pharmacology (4 works) · Angiogenesis (2 works) · Cancer Research (2 works) · Chemistry (2 works) · In vitro (2 works) · In vivo (2 works) · Internal Medicine (2 works)