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Jing-Yi Tang

Biographic Data

ID7280494
NAMEJing-Yi Tang
GIVEN NAMESJing-Yi
FAMILY NAMETang
SIGNATURETANG J
VERIFIEDNo
TOTAL WORKS2
TOTAL CITATIONS0
WORKS AS AUTHOR2
WORKS AS EDITOR0
FIRST PUBLICATION YEAR2017
LATEST PUBLICATION YEAR2022
H-INDEX0
  • Tribulus terrestris L. extract ameliorates atherosclerosis by inhibition of vascular smooth muscle cell proliferation in ApoE−/− mice and A7r5 cells via suppression of Akt/MEK/ERK signaling

    Open Access•Jing Zhang, Wai-Rong Zhao et al.•ARTICLE•Journal of Ethnopharmacology•2022

    FSEE presents potent anti- AS and VSMC proliferation activities and the underlying mechanism is likely to the suppression of Akt/MEK/ERK signaling. The active components of FSEE are JL-6 and its potential active pharmaceutical ingredients Tigo and Heco. So, FSEE and its active compounds may be potential therapeutic drug candidates for AS

  • Spatholobi Caulis extracts promote angiogenesis in Huvecs in vitro and in zebrafish embryos in vivo via up-regulation of VEGFRs

    Open Access•Zhong-Yan Zhou, Li-Yun Huan et al.•ARTICLE•Journal of Ethnopharmacology•2017

No prominent works on this page.

  • Spatholobi Caulis extracts promote angiogenesis in Huvecs in vitro and in zebrafish embryos in vivo via up-regulation of VEGFRs

    Open Access•Zhong-Yan Zhou, Li-Yun Huan et al.•ARTICLE•Journal of Ethnopharmacology•2017

  • Tribulus terrestris L. extract ameliorates atherosclerosis by inhibition of vascular smooth muscle cell proliferation in ApoE−/− mice and A7r5 cells via suppression of Akt/MEK/ERK signaling

    Open Access•Jing Zhang, Wai-Rong Zhao et al.•ARTICLE•Journal of Ethnopharmacology•2022

    FSEE presents potent anti- AS and VSMC proliferation activities and the underlying mechanism is likely to the suppression of Akt/MEK/ERK signaling. The active components of FSEE are JL-6 and its potential active pharmaceutical ingredients Tigo and Heco. So, FSEE and its active compounds may be potential therapeutic drug candidates for AS

Biochemistry (2 works) · Biology (2 works) · Cancer Research (2 works) · Medicine (2 works) · Pharmacology (2 works) · Angiogenesis (1 work) · Angiogenesis and VEGF in Cancer (1 work) · Atherosclerosis and Cardiovascular Diseases (1 work) · Cell Biology (1 work) · Cell growth (1 work)

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