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Christine Søholm Hansen

Biographic Data

ID7971435
NAMEChristine Søholm Hansen
GIVEN NAMESChristine Søholm
FAMILY NAMEHansen
SIGNATUREHANSEN C S
AFFILIATIONSStatens Serum Institut
ORCID0000-0002-7518-4663
VERIFIEDYes
TOTAL WORKS4
TOTAL CITATIONS0
AUTHOR COUNT4
EDITOR COUNT0
FIRST PUBLICATION YEAR2016
LATEST PUBLICATION YEAR2019
H-INDEX0
  • Discovery of the first genome-wide significant risk loci for attention deficit/hyperactivity disorder

    Open Access•Ditte Demontis, 23andMe Research Team et al.•ARTICLE•Nature Genetics•2019

    Attention deficit/hyperactivity disorder (ADHD) is a highly heritable childhood behavioral disorder affecting 5% of children and 2.5% of adults. Common genetic variants contribute substantially to ADHD susceptibility, but no variants have been robustly associated with ADHD. We report a genome-wide association meta-analysis of 20,183 individuals diagnosed with ADHD and 35,191 controls that identifies variants surpassing genome-wide significance in…

  • Identification of common genetic risk variants for autism spectrum disorder

    Open Access•John Grove, BUPGEN BUPGEN et al.•ARTICLE•Nature Genetics•2019

  • Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression

    Open Access•eQTLGen eQTLGen, Naomi R Wray et al.•ARTICLE•Nature Genetics•2018

  • DNA Methylation at the Neonatal State and at the Time of Diagnosis: Preliminary Support for an Association with the Estrogen Receptor 1, Gamma-Aminobutyric Acid B Receptor 1, and Myelin Oligodendrocyt…

    Open Access•Judith Becker Nissen, Christine Søholm Hansen et al.•ARTICLE•Frontiers in Psychiatry•2016

    Obsessive-compulsive disorder (OCD) is a neuropsychiatric disorder. Non-genetic factors and their interaction with genes have attracted increasing attention. Epigenetics is regarded an important interface between environmental signals and activation/repression of genomic responses. Epigenetic mechanisms have not previously been examined in OCD in children and adolescents. The aim of the present study was to examine the DNA methylation profile of …

No prominent works on this page.

  • DNA Methylation at the Neonatal State and at the Time of Diagnosis: Preliminary Support for an Association with the Estrogen Receptor 1, Gamma-Aminobutyric Acid B Receptor 1, and Myelin Oligodendrocyt…

    Open Access•Judith Becker Nissen, Christine Søholm Hansen et al.•ARTICLE•Frontiers in Psychiatry•2016

    Obsessive-compulsive disorder (OCD) is a neuropsychiatric disorder. Non-genetic factors and their interaction with genes have attracted increasing attention. Epigenetics is regarded an important interface between environmental signals and activation/repression of genomic responses. Epigenetic mechanisms have not previously been examined in OCD in children and adolescents. The aim of the present study was to examine the DNA methylation profile of …

  • Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression

    Open Access•eQTLGen eQTLGen, Naomi R Wray et al.•ARTICLE•Nature Genetics•2018

  • Discovery of the first genome-wide significant risk loci for attention deficit/hyperactivity disorder

    Open Access•Ditte Demontis, 23andMe Research Team et al.•ARTICLE•Nature Genetics•2019

    Attention deficit/hyperactivity disorder (ADHD) is a highly heritable childhood behavioral disorder affecting 5% of children and 2.5% of adults. Common genetic variants contribute substantially to ADHD susceptibility, but no variants have been robustly associated with ADHD. We report a genome-wide association meta-analysis of 20,183 individuals diagnosed with ADHD and 35,191 controls that identifies variants surpassing genome-wide significance in…

  • Identification of common genetic risk variants for autism spectrum disorder

    Open Access•John Grove, BUPGEN BUPGEN et al.•ARTICLE•Nature Genetics•2019

Biology (4 works) · Gene (4 works) · Genetic Associations and Epidemiology (3 works) · Genetics (3 works) · Genome-wide association study (3 works) · Genotype (3 works) · Psychology (3 works) · Single-nucleotide polymorphism (3 works) · Computational biology (2 works) · Genetic architecture (2 works)

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