Jonathan Mill
Biographic Data
| ID | 9508167 |
|---|---|
| NAME | Jonathan Mill |
| GIVEN NAMES | Jonathan |
| FAMILY NAME | Mill |
| SIGNATURE | MILL J |
| AFFILIATIONS | College of Medicine and Health University of Exeter Medical School Exeter UK |
| VERIFIED | No |
| TOTAL WORKS | 4 |
| TOTAL CITATIONS | 0 |
| AUTHOR COUNT | 4 |
| EDITOR COUNT | 0 |
| FIRST PUBLICATION YEAR | 2002 |
| LATEST PUBLICATION YEAR | 2023 |
| H-INDEX | 0 |
Genome‐wide DNA methylation analysis of aggressive behaviour: A longitudinal population‐based study
BACKGROUND: Human aggression is influenced by an interplay between genetic predisposition and experience across the life span. This interaction is thought to occur through epigenetic mechanisms, inducing differential gene expression, thereby moderating neuronal cell and circuit function, and thus shaping aggressive behaviour. METHODS: Genome-wide DNA methylation (DNAm) levels were measured in peripheral blood obtained from 95 individuals particip…
Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression
Influence of Life Stress on Depression: Moderation by a Polymorphism in the 5-HTT Gene
In a prospective-longitudinal study of a representative birth cohort, we tested why stressful experiences lead to depression in some people but not in others. A functional polymorphism in the promoter region of the serotonin transporter (5-HT T) gene was found to moderate the influence of stressful life events on depression. Individuals with one or two copies of the short allele of the 5-HT T promoter polymorphism exhibited more depressive sympto…
Role of Genotype in the Cycle of Violence in Maltreated Children
We studied a large sample of male children from birth to adulthood to determine why some children who are maltreated grow up to develop antisocial behavior, whereas others do not. A functional polymorphism in the gene encoding the neurotransmitter-metabolizing enzyme monoamine oxidase A ( MAOA ) was found to moderate the effect of maltreatment. Maltreated children with a genotype conferring high levels of MAOA expression were less likely to devel…
No prominent works on this page.
Role of Genotype in the Cycle of Violence in Maltreated Children
We studied a large sample of male children from birth to adulthood to determine why some children who are maltreated grow up to develop antisocial behavior, whereas others do not. A functional polymorphism in the gene encoding the neurotransmitter-metabolizing enzyme monoamine oxidase A ( MAOA ) was found to moderate the effect of maltreatment. Maltreated children with a genotype conferring high levels of MAOA expression were less likely to devel…
Influence of Life Stress on Depression: Moderation by a Polymorphism in the 5-HTT Gene
In a prospective-longitudinal study of a representative birth cohort, we tested why stressful experiences lead to depression in some people but not in others. A functional polymorphism in the promoter region of the serotonin transporter (5-HT T) gene was found to moderate the influence of stressful life events on depression. Individuals with one or two copies of the short allele of the 5-HT T promoter polymorphism exhibited more depressive sympto…
Genome-wide association analyses identify 44 risk variants and refine the genetic architecture of major depression
Genome‐wide DNA methylation analysis of aggressive behaviour: A longitudinal population‐based study
BACKGROUND: Human aggression is influenced by an interplay between genetic predisposition and experience across the life span. This interaction is thought to occur through epigenetic mechanisms, inducing differential gene expression, thereby moderating neuronal cell and circuit function, and thus shaping aggressive behaviour. METHODS: Genome-wide DNA methylation (DNAm) levels were measured in peripheral blood obtained from 95 individuals particip…
Biology (4 works) · Gene (4 works) · Genetics (4 works) · Genotype (4 works) · Psychology (4 works) · Medicine (3 works) · Child and Adolescent Psychosocial and Emotional Development (2 works) · Developmental psychology (2 works) · Genetic association (2 works) · Genetic Associations and Epidemiology (2 works)