Sarah Kittel-Schneider

Dados Biográficos

ID10409121
NOMESarah Kittel-Schneider
PRENOMESSarah
SOBRENOMEKittel-Schneider
ASSINATURAKITTEL-SCHNEIDER S
VERIFICADONão
TOTAL DE OBRAS7
TOTAL DE CITAÇÕES0
TOTAL COMO AUTOR7
TOTAL COMO EDITOR0
PRIMEIRO ANO DE PUBLICAÇÃO2012
ANO MAIS RECENTE DE PUBLICAÇÃO2024
ÍNDICE H0

  • The impact of emotional dysregulation and comorbid depressive symptoms on clinical features, brain arousal, and treatment response in adults with ADHD

    Open AccessJue Huang, Nicole Mauche et al.ARTICLEFrontiers in Psychiatry2024

    Our findings may support the assumption that ED may be an important feature of ADHD. The use of EEG-based brain arousal regulation as a diagnostic and predictive tool in ADHD in the presence of ED and comorbid depressive symptoms should be further investigated

  • The Quantified Behavioral Test—A Confirmatory Test in the Diagnostic Process of Adult ADHD

    Open AccessNathalie Brunkhorst-Kanaan, Moritz Verdenhalven et al.ARTICLEFrontiers in Psychiatry2020

    The differential diagnosis of attention deficit hyperactivity disorder (ADHD) in adulthood is complicated by comorbid disorders, but also by the overlapping of main symptoms such as inattentiveness, impulsivity, and hyperactivity with other disorders. Neuropsychological tests like continuous performance tests (CPT) try to solve this dilemma by objectively measurable parameters. We investigated in a cohort of n=114 patients presenting to an ADHD o…

  • Prevalence of Prediabetes and Diabetes Mellitus Type II in Bipolar Disorder

    Open AccessSarah Kittel‐Schneider, Sarah Kittel-Schneider et al.ARTICLEFrontiers in Psychiatry2020

    When controlled for BMI, in our study there was no significantly increased rate of T2D in BD. We thus suggest that overweight and obesity might be mediating the association between BD and diabetes. Underlying causes could be shared risk genes, medication effects, and lifestyle factors associated with depressive episodes. As the latter two can be modified, attention should be paid to weight changes in BD by monitoring and taking adequate measures …

  • Proteomic Profiling as a Diagnostic Biomarker for Discriminating Between Bipolar and Unipolar Depression

    Open AccessSarah Kittel‐Schneider, Sarah Kittel-Schneider et al.ARTICLEFrontiers in Psychiatry2020

    The results of this preliminary study suggest that future discrimination between bipolar and unipolar depression in a single case could be possible, using predictive biomarker models based on blood proteomic profiling

  • Impulsivity and Venturesomeness in an Adult ADHD Sample

    Open AccessO Grimm, Heike Weber et al.ARTICLEFrontiers in Psychiatry2020

    While impulsivity is a basic feature of attention-deficit/hyperactivity disorder (ADHD), no study explored the effect of different components of the Impulsiveness (Imp) and Venturesomeness (Vent) scale (IV7) on psychiatric comorbidities and an ADHD polygenic risk score (PRS). We used the IV7 self-report scale in an adult ADHD sample of 903 patients, 70% suffering from additional comorbid disorders, and in a subsample of 435 genotyped patients. Ve…

  • Analysis of the Influence of microRNAs in Lithium Response in Bipolar Disorder

    Open AccessCéline S Reinbold, Andreas J Forstner et al.ARTICLEFrontiers in Psychiatry2018

    Bipolar disorder (BD) is a common, highly heritable neuropsychiatric disease characterized by recurrent episodes of mania and depression. Lithium is the best-established long-term treatment for BD, even though individual response is highly variable. Evidence suggests that some of this variability has a genetic basis. This is supported by the largest genome-wide association study (GWAS) of lithium response to date conducted by the International Co…

  • Expression of Monoamine Transporters, Nitric Oxide Synthase 3, and Neurotrophin Genes in Antidepressant-Stimulated Astrocytes

    Open AccessSarah Kittel‐Schneider, Sarah Kittel-Schneider et al.ARTICLEFrontiers in Psychiatry2012

    These data provide further evidence for a role of astroglial cells in the molecular mechanisms of action of antidepressants