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Discrimination, segregation, and chronic inflammation

Testing the weathering explanation for the poor health of Black Americans

Bibliographic Data

ID9785960
AuthorsR L Simons (0000-0002-4902-0462, University of Georgia, corresponding author), Man-Kit Lei (0000-0002-7757-6548, University of Georgia), Steven R Beach (0000-0001-8053-7761, University of Georgia), Steven R H Beach, A B Barr (0000-0003-3105-2703, University at Buffalo, State University of New York), Leslie Gordon Simons (0000-0001-8962-7456, University of Georgia), Frederick X Gibbons (0000-0003-1205-1481, University of Connecticut), Robert A Philibert (0000-0001-7822-4977, University of Iowa)
Year2018
Volume54
Issue10
Pages1993-2006
Publication date2018-10-01
Peer ReviewedYes
Open AccessYes
TypeARTICLE
VenueDevelopmental Psychology (JOURNAL)
Journal identifiersISSN: 0012-1649 • E-ISSN: 1939-0599
PublisherAmerican Psychological Association (APA) (PUBLISHER)
DOI10.1037/dev0000511
PMID30234347
OpenAlexW2890792344
LanguageEN
Citations received44
References cited40

Several studies have reported a relation between race-related stressors and the poor health of Black Americans. Such findings raise questions regarding the mediating biological mechanisms that might account for this link. The present study investigated elevated systemic inflammation, a factor shown to be a strong predictor of chronic illness and mortality in all ethnic populations, as a possible factor. Using 7 waves of data from the Family and Community Health Study, collected over a 20-year period from over 400 Black Americans, we investigated the extent to which exposure to discrimination and segregation at various points in the life course predicted adult inflammation at age 28. Our analyses examined whether cumulative stress, stress generation, or predictive adaptive response (PAR) models best accounted for any associations that existed between these race-related stressors and adult inflammation. At every wave of data collection, assessments of discrimination and segregation were related to adult inflammation. However, multivariate analyses using structure equation modeling indicated that the PAR model best explained the effect of these race-related stressors on inflammation. Exposure to discrimination and segregation during the juvenile years predicted adult inflammation and amplified the inflammatory effect of adult exposure to these race-related stressors. These effects were considerably more robust than that of traditional health risk factors such as diet, exercise, smoking, and low SES. Implications of these findings are discussed, including the limitations of the widely accepted risk factor approach to increasing the health of Black Americans. (PsycINFO Database Record

Allostatic load · Biology · Developmental psychology · Ethnic group · Inflammation · MEDLINE · PsycINFO · Race (biology · Risk factor · Stressor · Clinical Psychology · Demography · Health disparities and outcomes · Immunology · Internal Medicine · Medicine · Psychology · Racial and Ethnic Identity Research · Stress Responses and Cortisol · Gerontology

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Unique citing works44
Citations per year6,29
Citation span2019 - 2026 (8)
Citation velocitycurrent
Highly citedNo
Citation typesNeutral: 44

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