Zhaoyong Li
Biographic Data
| ID | 7269383 |
|---|---|
| NAME | Zhaoyong Li |
| GIVEN NAMES | Zhaoyong |
| FAMILY NAME | Li |
| SIGNATURE | LI Z |
| AFFILIATIONS | First Affiliated Hospital of Hunan University of Traditional Chinese Medicine |
| ORCID | 0000-0002-1502-4027 |
| VERIFIED | No |
| TOTAL WORKS | 3 |
| TOTAL CITATIONS | 0 |
| AUTHOR COUNT | 3 |
| EDITOR COUNT | 0 |
| FIRST PUBLICATION YEAR | 2023 |
| LATEST PUBLICATION YEAR | 2026 |
| H-INDEX | 0 |
Dingxin recipe III ameliorates endothelial cell senescence in atherosclerosis by activating the Foxo3a/Pink1/Parkin axis
Our findings for the first time demonstrated that DXRIII effectively alleviates endothelial senescence and HFD-induced AS, possibly by activating FOXO3a and subsequently enhancing PINK1/Parkin-mediated mitophagy
BSHXF-medicated serum combined with ADSCs regulates the TGF-β1/Smad pathway to repair oxidatively damaged NPCs and its component analysis
By regulating the TGF-β1/Smad pathway, BSHXF-medicated serum promoted ADSCs to NPCs, effectively alleviated the cycle blockage of NPCs after oxidative damage, encouraged the growth and proliferation of NPCs, delayed the aging of NPCs, improved the deteriorating microenvironment around NPCs, and repaired oxidatively damaged NPCs. The combination of BSHXF or its compounds with ADSCs has great potential for the treatment of IDD in the future
Geniposide ameliorates atherosclerosis by regulating macrophage polarization via perivascular adipocyte-derived CXCL14
In summary, our findings suggest that geniposide protects ApoE -/- mice against WD-induced atherosclerosis by inducing M2 polarization of plaque macrophages via enhanced expression of CXCL14 in PVAT. These data provide novel insights into PVAT paracrine function in atherosclerosis and reaffirm geniposide as a therapeutic drug candidate for atherosclerosis treatment
No prominent works on this page.
BSHXF-medicated serum combined with ADSCs regulates the TGF-β1/Smad pathway to repair oxidatively damaged NPCs and its component analysis
By regulating the TGF-β1/Smad pathway, BSHXF-medicated serum promoted ADSCs to NPCs, effectively alleviated the cycle blockage of NPCs after oxidative damage, encouraged the growth and proliferation of NPCs, delayed the aging of NPCs, improved the deteriorating microenvironment around NPCs, and repaired oxidatively damaged NPCs. The combination of BSHXF or its compounds with ADSCs has great potential for the treatment of IDD in the future
Geniposide ameliorates atherosclerosis by regulating macrophage polarization via perivascular adipocyte-derived CXCL14
In summary, our findings suggest that geniposide protects ApoE -/- mice against WD-induced atherosclerosis by inducing M2 polarization of plaque macrophages via enhanced expression of CXCL14 in PVAT. These data provide novel insights into PVAT paracrine function in atherosclerosis and reaffirm geniposide as a therapeutic drug candidate for atherosclerosis treatment
Dingxin recipe III ameliorates endothelial cell senescence in atherosclerosis by activating the Foxo3a/Pink1/Parkin axis
Our findings for the first time demonstrated that DXRIII effectively alleviates endothelial senescence and HFD-induced AS, possibly by activating FOXO3a and subsequently enhancing PINK1/Parkin-mediated mitophagy
Internal Medicine (3 works) · Medicine (3 works) · Pharmacology (3 works) · Atherosclerosis and Cardiovascular Diseases (2 works) · Chemistry (2 works) · Adipocyte (1 works) · Adipokines, Inflammation, and Metabolic Diseases (1 works) · Adipose tissue (1 works) · Biochemistry (1 works) · Cancer Research (1 works)